Insulin- Wise Eating

Majid Ali, M.D.

Insulin-Wise Eating

For Improving Insulin Efficiency


Insulin-Wise Eating

For Improving Insulin Efficiency

Updated Jan. 16, 2018

Majid Ali, M.D.

 

To reverse pre-diabetes and diabetes (completely or partially), my primary objective is to lower both blood sugar and insulin levels by making insulin work better. For individuals with pre-diabetes with insulin toxicity but without high blood sugar levels, my goal is to lower blood insulin levels by increasing insulin efficiency.

Dr. Ali’s Insulin Normalization Plan

My Insulin Reduction Protocol has three components:

  1. A plan of food choices to prevent sugar spikes that trigger insulin spikes, and
  2. A plan to do daily gentle bowel and liver detox.
  3. A Plan for Spiritual Surrender

In the Table 2 below, I present a case study to show how blood glucose and insulin levels were lowered (by increasing insulin efficiency) with the clinical application of Dr. Ali’s Insulin Reduction Protocol. I follow this with some explanatory comments. In Table 1, I present the insulin and glucose values of an individual in good metabolic health.

 

Table 1. Insulin-conserving Profile of a 77-Yr-Old Metabolically Fit 5′ 5″ Man Weighing 133 Lbs. He Was Seen for Allergy Treatment.
6.23. 2010 Fasting 1 Hr 2 Hr 3 Hr
Insulin <2 24 29 30
Glucose 78 96 75 71

 

Table 2. Concurrent Reduction of Blood Insulin and Blood Sugar Levels With Dr. Ali’s Insulin Reduction Protocol in a 58-Yr-Old Woman With Complete Loss of Hair (Alopecia), Chronic Fatigue, Memory Deficit, Underactive Thyroid Gland, Allergy, and Mood Swings.
10.28.10 Fasting 1 Hr 2 Hr 3 Hr
Insulin 9.7 184.4 35.3 24
Glucose 102 133 79 73
11.23.1202
Insulin 12.7 87.7 50.2  
Glucose 96 117 77  

Diabetes Reversal Requires a Philosophy of Healing

Diabetes Type 2 can be reversed neither with the denial of dieting nor with euphoria of eating. Diabetes can be reversed only with a philosophy of eating and living. It requires knowing the difference between being “diabetes-literate” and “healing-literate.” Diabetes is the number one cause of blindness, neuropathy, toe and limb amputations, kidney failure leading to dialysis, and increased risk of strokes, memory loss, and heart attacks. So reversing diabetes is an act of self-compassion. If these words pull you toward making an honest attempt to lose diabetes Type 2, please consider studying “Dr. Ali’s Course on Healing” (available at www.aliacademy.com).


 

Five Important Facts About Insulin-Wise and Insulin-Unwise Foods

I coined the terms Insulin-Wise and Insulin-Unwise Foods to raise consciousness about the serious adverse effects of insulin spikes on all cell populations in the body.

  1. Insulin in excess (insulin toxicity) is fattening and inflaming.
  2. Healthy fats are insulin-friendly.
  3. Healthy proteins are insulin-friendly.
  4. All breads, pastas, fruits and fruit juices, and sugars are insulin-unfriendly.
  5. Vegetables are insulin-friendly.

Simply stated, Insulin-Wise foods facilitate insulin signals and other functions, while Insulin-Unwise foods impede or block insulin signals and other functions. I present this subject at length in my book entitled “Dr. Ali’s Plan for Reversing Diabetes” and in a 40-minute video seminar that can be downloaded from www.aliacademy.org or by calling 1-800-633-6226.

Below are guidelines for Dr. Ali’s Insulin Diet. If initially this diet plan is found to be too hard and restrictive, one or two days a week may be taken as free days. Of course, some insulin benefits will be lost on such days.

Insulin-Friendly Breakfast for Saving Insulin and Preventing Diabetes

  1. Please consider “Dr. Ali’s Breakfast” (protein shake described below) four or five days a week.

2.` Take eggs and vegetables for breakfast the remaining two days.

  1. No fruit juices, breads, or toast.

Insulin-Friendly Lunch for Saving Insulin and Preventing Diabetes

  1. Large salad with goat cheese, chicken, or fish. All the olive or sesame oil you wish.
  2. Uncooked, steamed, or lightly stir-fried vegetables. All the olive or sesame oil you wish.

Mid-Afternoon Snack

Use four to six ounces of the Dr. Ali’s Breakfast shake (prepared in the morning and carried to work).

Insulin-Friendly Dinner for Saving Insulin and Preventing Diabetes

  1. All healthy fats and oils
  2. All healthy proteins
  3. Uncooked, steamed, or lightly stir-fried vegetables with melted butter or olive oil (to your heart’s content).

Optimal Breakfast Choices for Diabetes

Dr. Ali’s Breakfast on five to six days per week comprising: (1) two tablespoons of a protein powder containing 85 to 90 percent calories in proteins and peptides; (2) two tablespoons of a granular lecithin; (3) two tablespoons of freshly ground flaxseed (the use of a coffee grinder is recommended); (4) 12 to 16 ounces of organic vegetable juice (avoiding or minimizing the use of carrots and red beets); and (5) 12 to 16 ounces of water. A few ounces of seltzer water or a few drops of lemon juice may be added to suit personal taste. I personally consume this mixture in portions of six to eight ounces with my morning nutrient and herbal protocols during the period of my morning exercise, meditation, and preparation for work. I have not yet encountered any negative impact of the protein content in this breakfast on renal function. Still, individuals with serum creatinine levels above the normal range need to be monitored for renal function.

Insulin Channel on YouTube Science, Health, and Healing Encyclopedia

I offer about 75 videos on insulin in health and disease on my my YouTube Science, Health, and Healing Encyclopedia. I especially recommend the channel entitled “Seven Faces of Insulin Toxicity”: http://www.youtube.com/watch?v=zxtVhe0mnf4

Insulin – the Minister of Energy and Metabolism

I designate insulin to be the Minister of Energy and Metabolism to the Oxygen king of the human body. By its signals, it regulates the energy of all cells in the body. Of necessity, this means that insulin has a role to play in the health preservation of all such cells.

Insulin is a hormone produced in specialized cells of the pancreas gland called beta cells. It is a string made up of 51 amino acid molecules and has a molecular weight of 5808 Daltons. Insulin performs diverse metabolic and non-metabolic functions in the body. As for metabolism, its major functions include the transfer of glucose from the blood into the liver and muscle cells for storage and into the fatty tissues to stop the use of fat as fuel. Among the major non-metabolic functions are its roles in cellular development, differentiation, and death.

Related Articles

 Oxygen Homeostasis and Oxygen Models of Diseases

* Insulin Homeostasis and Diabetes

* Insulin-Wise Foods, Insulin-Saving Recipes

* Dr. Ali’s Insulin Reduction Protocol – For Improving Insulin Efficiency

* Dr. Ali’s Insulin-Wise Breakfast

* Dr. Ali’s Insulin-Wise Breakfast – Personalized

* Insulin-saving Tuna-Tiki

* Insulin-saving Vege-Tiki

* Insulin-saving Palak-Tiki

* Insulin-saving Almond snack

The Diabetes Question

Majid Ali, M.D.

Does Diabetes Begin As a Rising Blood Sugar Disease Or As a Rising blood Insulin Disease?

We Will Let Call It  

The Diabetes Question.


If the answer to the diabetes question is that it begins with rising blood insulin levels,  not with rising blood glucose levels, then the following new questions arise?

Question: Is excess insulin (hyperinsulinism) toxic to the body organs?

Answer, Yes, Excess insulin is fattening, fermentiing, and inflaming. It swells the liver and shrinks the brain. It is pro-cancer, pro-inflammation, and pro-degenerative diseases. In damages endo cells which lines the inside of the entire cardiovascular system and affects blood circulation everywhere in the body. Simply stated, excess insulin (insulin toxicity) is “pro-accelerated pro-aging.” 

Question: Can insulin toxicity be assessed with blood sugar tests?

Answer. No.

Question: In most people, how long does insulin toxicity go on undetected before blood sugar levels rise enough to make diabetes diagnosable with blood sugar tests?

Answer, for five, ten, or more years?

Question: Do doctors usually always test for blood insulin level before they test for blood sugar level?

Answer, No.


 

What Must Be Known About Crucial Diabetes and Its Complications

(In this article the terms diabetes and Type 2 dabetes are used interchangeably)

Diabetes (Type 2 Diabetes,T2D) Cannot Be Diagnosed In Time Without Insulin Tests, Diabetes Cannot Be Reversed Without Insulin Intelligence. Nor Can Diabetes Complications Be Prevented or Reversed Without Insulin Intelligence.


 

Summary

Diabetes Is Not a Sugar Problem,

It Is a Problem of Insulin Toxicity (Hyperinsulinism).

Insulin Toxicity Predates Diabetes by Five, Ten, or more Years, and Directly Leads to the Disease.


 

The Cost of Neglected Insulin Testing 

Hyperinsulinism (insulin toxicity) inflicts cellular injury in nearly all cellular populations in the body.  During the  years insulin toxicity remains ,undetected and untreated, simply because insulin testing is neglected by practitioners. Why?

Blood insulin testing is not considered a standard of care by those who control $1.3 trillion yearly spending for medical care in the United States. After considering the evidence I present in this and other articles in my “Diabetes Question Series,” the readers will decide for themselves as to the real reason for neglected insulin testing.  

I Leave the answer to readers.    


What Is Insulin Intelligence?

Simply stated, excess insulin (insulin toxicity and hyperinsulinism by other names)  is a fire which burns all parts of the body. It may start in different places and spread differently but the end result is always shortened life span with different diseases.

A practitioner who answers this questions with the “diabetes-hyperinsulinism” prevailing view does not, in my opinion, serve his patients well. Anyone who answers the question with one-liners recognizing insulin as the “life-span” hormone without does not deserve anyone’s time. As for me, I want to invite you to consider these questions by taking my free-of-cost course at this web site. A library of my selected article, published papers, and short videos is included in this post. Readers interested in my book on reversing diabetes and video seminar downloads can access these materials at http://www.aliacademy.org.


 

The Diabetes Question:

Can insulin regulation be assesses with sugar tests?

Specifically, can diabetes be detected in time with fasting blood sugar test, A1c blood tests, two-hour post prandial (after a meal) blood sugar level?

The answer: Categorically not.


 

What Is Optimal Insulin Homeostasis?

First, when the blood insulin levels after a glucose challenge are quite low;

Second, blood glucose after a glucose challenge are within low physiologic range.

Third, when there is no history of diabetes in parents and grandparents.

Fourth, when there is no insulin toxicity.

Fifth, when the immune system is robust and there is no chronic . immune-inflammatory disease.

Question: Can one optimize one’s insulin homeostasis? One can only answer this question for oneself.


 

One can tell oneself lies, but nature does not grant permission to believe one’s own lies. 


Can insulin homeostasis (insulin regulation as a whole) be assessed with blood sugar tolerance  test, A1c blood tests, two-hour post prandial (after a meal) blood glucose tests, as for instance the tolerance test done for gestational diabetes?

The answer: Categorically not.


To provide a broader context for due deliberation of the above questions, please consider sets of blood insulin and glucose profiles below which were prepared with fasting and timed post-glucose challenge.
       Table  1  Control Profiles
       Table 2,3 Blood glicose tests are inappropriate for assessing insulin homestasis
       Table 4.  Hyperinsulinism in Autism Spectrum Disorder 

Table 1. Two Sets of Control Insulin and Glucose Profiles

1.Healthy control subject:. Case 1.

                 INSULIN :    <2 uIU/mL, 18, uIU/mL, 4,       and <2;    

                 GLUCOSE:    77, 168, 109, 74, 52.

2. Healthy Control Subject: Case 2  

               INSULIN :    3 uIU/mL, 11, uIU/mL, 7,   and <2;    

               GLUCOSE:    81  157, 98, 63, 52.


The Challenge in Reversing Diabetes

is not to know what any doctor thinks about diabetes and drugs used to treat diabetes but how to learn to think for yourself about insulin, health, and healing.

I suggest you spend time at http://www.alidiabetes.org 


The Most Important Question in the Prevention and Reversal of Diabetes (Type 2)

No question is more important for stemming the global tides of insulin toxicity and diabetes than the question in the title of this post.


The Answer:

Insulin levels rise first, usually by five, ten, or more years before blood sugars level rise.
Why is this important?
Because insulin toxicity continues to cause cellular damage in the liver, kidneys, heart, brain, eyes and other organs unknown to the patient and the doctor if insulin tests are not done. For more info, go to http://www.Ali Diabetes.Org for the author’s free-access course at
http://www.Ali Diabetes.Org.

https://wordpress.com/post/alidiabetes.org/2966


Table 2. Insulin Homeostasis Categories in 506 Study Subjects Without Type 2 Diabetes
Insulin Category*
Percentage of Subgroup
Mean Peak Glucose  mg/dL
(mmol/mL)
Mean Peak Insulin (uIU/mL)
Exceptional Insulin Homeostasis.N 12**
1.7%
110.2     (6.12)
14.3
Optimal Insulin Homeostasis N =126
24.9 %
121.2     (6.73)
26.7
Hyperinsulinism, Mild                N =197
38.9 %
136.5   (7.58)
58.5
Hyperinsulinism,  Moderate       N =134
26.5 %
147.0    (8.16)
109.1
Hyperinsulinism,  Severe             N =  49
9.7 %
150.0    (8.33)
(less than time and a half higher) 
231.0
(nearly 17 times higher)
#   Correlation coefficient, r value, for means of peak glucose and insulin levels in the five insulin categories is 0.84.
  *Criteria for classification: (1) Exceptional insulin homeostasis, a subgroup of optimal insulin homeostasis with fasting insulin concentration of <2 uIU/mL and mean peak insulin concentration of <20; (2) optimal insulin homeostasis, peak insulin <40 accompanied by unimpaired glucose tolerance; (3) mild insulin homeostasis, peak insulin  between <40 and 80 uU/mL;  accompanied by unimpaired glucose tolerance; ; (3) moderate insulin homeostasis, peak insulin  between <80 uU/mL and 160 uIU/mL accompanied by unimpaired glucose tolerance;  and (4) severe insulin homeostasis, peak insulin  > 160 uU/mL accompanied by unimpaired glucose tolerance.

Why Do Diabetics Need Insulin Shots?

Because Their Pancreas Has Exhausted Its Lifetime Capacity of Produce Sufficient Insulin

Note the extremely high blood insulin level (298 uIU/mL) still cannot keep the blood glucose level in the normal non-diabetic level.
Table 3. Insulin Homeostasis Categories in 178 Study Subjects With Type 2 Diabetes.
Insulin Category
Percentage of Subgroup
Mean Peak Glucose, mg/dL
(mmol/mL)
Mean Peak Insulin (uIU/mL)
Diabetic Hyperinsulinism, Mild              N =  53
29.0%
252.0   (14.00)
55.4
Diabetic Hyperinsulinism, Moderate    N =  42
24.0%
242.1   (13.45)
112.4
Diabetic Hyperinsulinism, Severe          N =  24
13.9%
224.6   (12.47)
298.0
Diabetic  Insulin Deficit                             N =  59
33.1%
294.0    (16.33)
22.9

What Is Optimal Insulin Homeostasis?

It is the lowest blood insulin levels that can keep the blood glucose levels in the normal range.
In other words, It is ideal state of insulin utilization, in which insulin toxicity does not exist, nor is insulin wasted because there is too much of it in the blood.
is not wasted .
In 2017, in a large survey of insulin and glucose profiles in the general New York metropolitan population, my colleagues and I reported a hyperinsulinism prevalence of 75.1%. Below is the link to get free access to the full text of this report:

http://www.townsendletter.com/Jan2017/insulin0117.html

Or, you may get the report on this website by entering , please use the the following words on the search box of the site:  “Shifting Focus from Glycemic Status.”

Examples of Insulin and Glucose Profiles of Individuals With Perfect Insulin Regulation

Table 1. Post-Glucose Load Insulin and Glucose Profiles of Seven Individuals With Optimal Insulin Homeostasis as Defined Above.
Fasting
½-Hr
1-Hr
2-Hr
3-hr
Insulin Profile 1. Insulin And Glucose Profiles of a 47-yr-old 5′ 5″ Male Runner Weighing 130 lbs. Who Presented With Inhalant Allergy and Hemorrhoids.
Insulin uIU/mL
1.5
9.7
9.0
4.6
<1.0
Glucose mg/dL
72
148
134
108
54
Insulin Profile 2. Insulin and Glucose Profiles of a  45-Yr-Old  5’9″Man Weighing 125 lbs. Presenting With Allergy and Dry Skin.
Insulin uIU/mL
1.0
2.7
9.8
2.7
<1.0
Glucose mg/dL
85
110
75
70
52
Insulin Profile 3. Insulin and Glucose Profiles of a 51-year-old 5’6″ Man Weighing 120 lbs. He Consulted Me for Cardiac Rhythm Disorder, Hypothyroidism and  Allergy.
Insulin uIU/mL
2.9
6.0
11.5
2.5
Glucose mg/dL
89
103
134
110
59
Insulin Profile 4. Insulin and Glucose Profiles of a 52-Yr-Old 5’1″ Woman Weighing 120 lbs. Presenting With Constipation and  Allergy.
Insulin uIU/mL
<2
17
15
6
Glucose mg/dL
78
61
72
71
Insulin Profile 5. Insulin and Glucose Profiles of a  52-Yr-Old 5’ 7″ Man Weighing 155 lbs. Presenting With Anxiety, Depression, and Diarrhea. A1c. 5.3%
Insulin uIU/mL
2.0
8.1
19.6
17.7
4
Glucose mg/dL
94
140
158
91
73
Insulin Profile 6. Insulin and Glucose Profiles of a  62-Yr-Old  5’3″ Woman Weighing 114 lbs. Presenting With Allergy and Hand Arthralgia.
Insulin uIU/mL
1.8
17.8
11.0
10.0
Glucose mg/dL
80
159
76
75
68
Insulin Profile 7. Insulin and Glucose Profiles of a 51-year-old 5’2″ Woman Weighing 120 lbs. She Consulted Me for Hypothyroidism and  Allergy
Insulin uIU/mL
3.2
11.8
2.4
1.9
Glucose mg/dL
86
110
75
70
52
Insulin Lab Reference Ranges Not  Fit for Use
In a previous report the author and his colleagues have highlighted the serious problem of inappropriate prevailing reference ranges for blood insulin concentrations.13 The data in Table 2 reproduced from that publication dramatically illustrates the dimension of this problem with findings of a survey of major laboratories in the New York City metropolitan area. The study data also calls into question the clinical value of single and random blood insulin test results. Photographs of illustrative lab reports are posted online.14

Absurd Laboratory Reference Ranges

Table 2. Upper and Lower Limits of Laboratory Insulin Reference  Ranges Expressed In uIU/mL Following a Standard Glucose Load From Six Major Clinical Laboratories in the New York Metropolitan Area.2
Laboratory
Fasting
1 Hr
2 Hr
3 Hr
Laboratory 1
1.9 – 23
8  –  112
5 – 35
Laboratory  2
2.6 – 24.9
0.0  – 121.9
0.0 – 163.5
Laboratory  3
2.6 – 24.9
8  –  112
5  –  55
3  –  20
Laboratory  4
6  – 27
20  –  120
18  –  56
8  –  22
Laboratory  5
00  – 30
30  –  200
40  – 300
50  – 150
Laboratory  6
Does not include insulin ranges in the report. Instead it includes the following note: Insulin analogues may demonstrate non-linear cross-reactivity in this essay. Interpret results accordingly. Personal communications with clinicians revealed that they do not find this laboratory note to be helpful.
 
 

Spectrum of Insulin Dysfunction and Hyperinsulinism in Autism

Table 4 presents insulin and glucose profiles of 10 patients with dysautonomia. Note that all patients suffered from allergic disorders.
Table 4. Insulin and Glucose Profiles of Individuals With Autism.
The Blood Insulin and Glucose Levels Are Expressed in uIU/mL and mg/dL respectively.
Fasting
½ Hr
1 Hr
2 Hr
3 Hr
Autism Case 1. Insulin and Glucose Profiles of 14-Yr-Old 5’ 9” Boy Weighing 115 lbs.Who Presented Without Expressive Speech Since Birth.
Insulin uIU/mL
24
300
235
211
83
Glucose mg/dL
83
129
98
95
61
Autism Case 2. Insulin Profile and Glucose Profiles of 15-Yr-Old Boy With  Autism, Allergy, and Fatigue.
Insulin uIU/mL
10.4
43.7
37.6
33.7
7.8
Glucose mg/dL
79
104
86
82
53
Autism Case 3. Insulin and Glucose Profiles of 17-Yr-Old-Boy With Autism, Eczema, And Anxiety.
Insulin uIU/mL
24.4
N/A
73.8
71.6
28.0
Glucose mg/dL
95
N/A
79
79
69
Autism Case 4.  Insulin and Glucose Profiles of 8-Yr-Old Boy Presenting With Autism, Sudden Mood Shifts, and Inhalant Allergy.
Insulin uIU/mL
6.2
40.36
41.5
24.8
3.9
Glucose mg/dL
96
192
131
109
57
Autism Case 5. Insulin and Glucose Profiles of A Three-Year-Old  Boy With Asperger’s Syndrome, Temper Tantrums, Eczema, And Inhalant Allergy.
Insulin uIU/mL
1.28
14.3
0.33
Glucose mg/dL
71
126
88
Autism Case  6. Insulin and Glucose Profiles Of A Four-Year-Old Boy Weighing 35 lbs. Limited expressive speech, Often in non-communicative trance. Mother’s Words: “Very Intelligent In Things That Interest Him.”
Insulin uIU/mL
2.3
24.2
20.2
17.8
0.8
Glucose mg/dL
89
151
102
98
79
Autism Case 7 .  Insulin and Glucose Profiles of A 5-yr-old Boy With Autism Focus Disorder. No Expressive Speech Until Age 30 Months, Single Words 10-15 Words. No Voluntary Sentences. Eczema, Recurrent Ear Infections.
Insulin uIU/mL
1.31
47.16
43.99
Glucose mg/dL
64
127
150
Autism Case 8 . Insulin And Glucose Profiles  of  A 7-Yr-Old Boy Presenting With Diagnoses of Autism, Inhalant Eczema, Food Allergy, and History of Multiple Courses of Antibiotics for Sore Throats.
Insulin uIU/mL
11.0
Glucose mg/dL
73
Autism Case 9. Insulin And Glucose Profiles  of A Six-Yr-Old Boy Presented With Autism, Hypothyroidism, Food and Inhalant Allergy.
Insulin uIU/mL
13.0
Glucose mg/dL
85
The staff of a university hospital mishandled the blood samples on two different occasions.
Autism Case 10. Insulin and Glucose Profile of A 28-yr-old Man Who Was Diagnosed With Autism with complete Absence of Expressive Speech Until Age 4 And Then Transitioned to Asperger’s Syndrome. At Age 21, He Was An Excellent Athlete But Could Speak Only To His Mother.
Insulin uIU/mL
7
174
365
71.9
7.9
Glucose mg/dL
81
178
160
85
56
Follow-Up Testing One Year Later
Insulin uIU/mL
8.2
139.9
152.0
40.82
2.82
Glucose mg/dL
88
128
125
100
47

Free-Access Library for Reversing Diabetes.

First things first: Only you can reverse your diabetes, not anyone else.

What Comes First Rising Blood Sugar Level, Or Rising Blood Insulin Level?

 

Majid Ali, M.D.

No question is more important for stemming the global tides of insulin toxicity and diabetes than the question in the title.

(Part of the Diabetes Question Series)


 

The Answer:

Insulin levels rise first, usually by five, ten, or more years before blood sugars level rise.

Why is this important?

Because insulin toxicity continues to cause cellular damage in the liver, kidneys, heart, brain, eyes and other organs unknown to the patient and the doctor if insulin tests are not done.


 

Insulin Toxicity, Silent Stroke, Brain Lacuna, and Coming Years

Majid Ali, M.D.

A brain lacuna is a localized area of loss of brain cells which does not cause any signs and symptoms of brain damage. It is considered as a small silent stroke. It must also be considered an important wake-up call for gardener thinking.


A Bit of Gardener Thinking for Brain Health

 

As roots are to roses, so the liver to the brain.

As roots are to roses, so the the bowel to the liver.


 

Gardener Thinking

Gardeners are usually clear-headed. They don’t count wilting leaves and classify diseases by the number, form, and location of wilting leaves. They think of the health of the soil and sunlight for their flowering plants.


 

For bowel health, please consider my videos and articles under the Seed-Feed-Weed category.

Selected Articles and Videos

Hyperinsulinism (insulin toxicity) and stroke | The Ali Academy Community

Citations for the Diabetes Question Series

MAJID ALI, M.D.

Free Access Library of Articles for Reversing Hyperinsulinism and Type 2 Diabetes

(Part of the Diabetes Question Series)


References 
1.     M. Respiratory-to-Fermentative (RTF) Shift in ATP Production in Chronic Energy Deficit Disorders. Townsend Letter for Doctors and Patients. 2004;253:64-65.
2.     Ali M. Oxygen and Aging. Book Ali M. Oxygen and Aging. (Ist ed.) New York, Canary 21 Press. Aging Healthfully Book 2000. .
3.     Ali M. Succinate Retention. In: Chouchani ET, Victoria R. Pell VR, Edoardo Gaude E, et. al. Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS. Nature. 2014;515:431–435.
4.     Ali M. Succinate Retention: The Core Krebs Dysfunction in Immune-Inflammatory Disorders. Townsend Letter. 2015;388:84-85.
5.     Ali M. Epidemic of Dysoxygenosis and the Metabolic Syndrome. In: The Principles and Practice of Integrative Medicine. Volume 5. Pp 246-256. Canary 21 Press. New York. 2005.
6.     Ali M. Dysox and Climatic Chaos –  The primacy of oxygen issues over carbon issues. Part I. Townsend Letter-The examiner of Alternative Medicine. 2008;299:125-132.
7.     Ali M. Oxygen, Insulin Toxicity, Inflammation, And  the Clinical Benefits of Chelation. Part I. Townsend Letter-The examiner of Alternative Medicine. 2009;315:105-109. October, 2009.
8.     Ali M. Insulin Reduction and EDTA Chelation: Two Potent and Complementary Approaches For Preventing and Reversing Coronary Disease. Oxygen, Insulin Toxicity, Inflammation, and the  Clinical Benefits of Chelation – Part II. Townsend Letter-The examiner of Alternative Medicine. 2010;323:74-79. June 2010.
9.     Ali M. Dysox Model of Diabetes and De-Diabetization Potential. Townsend Letter-The examiner of Alternative Medicine. 2007; 286:137-145.
10. Ali M. Plan for Reversing Diabetes. New York, Canary 21 Press. Aging Healthfully Book 2011.
11. Ali M. Importance of Subtyping Diabetes Type 2 Into Diabetes Type 2A and Diabetes Type 2B. Townsend Letter-The Examiner of Alternative Medicine. 2014; 369:56-58.
12. Ali M. Dasoju S, Karim N, Amin J, Chaudary D. Study of Responses to Carbohydrates and Non-carbohydrate Challenges In Insulin-Based Care of Metabolic Disorders.  Townsend Letter-The Examiner of Alternative Medicine. 2016; 391:48-51.
13. Ali M, Fayemi AO, Ali O. Dasoju S, et al. Shifting Focus From Glycemic Status to Insulin Homeostasis. .  Townsend Letter-The Examiner of Alternative Medicine. 2017;402:91-96.
14. Itoh Y, Kawamata Y, Harada M, et al. Free fatty acids regulate insulin secretion from pancreatic Description: eta cells through GPR40Nature;422:173–176.
15. Kahn SE, 1, Hull RL, Utzschneider KM. Mechanisms linking obesity to insulin resistance and type 2 diabetes. Nature 2006;444, 840-846.
16. Reaven GM, Hollenbeck C, Jeng CY, et al. Measurement of plasma glucose, free fatty acid, lactate, and insulin for 24 h in patients with NIDDMDiabetes. 1988;371020–1024.
17. Sako, Y. & Grill, V. E. A 48-hour lipid infusion in the rat time-dependently inhibits glucose-induced insulin secretion and B cell oxidation through a process likely coupled to fatty acid oxidationEndocrinology 127, 1580–1589 (1990). |
18. Rhodes, C. J. Type 2 diabetes — a matter of Description: eta-cell life and death? Science. 2005;307:380–384.
19. Kahn, S. E., Bergman, R. N., Schwartz, M. W., Taborsky, G. J. & Porte, D. Short-term hyperglycemia and hyperinsulinemia improve insulin action but do not alter glucose action in normal humansAm. J. Physiol.1992;262:E518–E523.
20. Ali  M. Molecular Basis of Autism and Dysuatonomia – The Impeded Progenitor Cell Progression (IPCP) model of ASD and Dysautonomia.  Townsend Letter for Doctors and Patients. 2017 (In press)
21. Ali  M.  Insulin Laboratory Ranges. https://alidiabetes.org/2016/02/25/insulin-laboratory-ranges/
22. Shoelson SE, Lee J, Goldfine AB. Inflammation and insulin resistance. J Clin Invest. 2006;116 :1793B1801.
23. Shulman G. Ectopic Fat in Insulin Resistance, Dyslipidemia, and Cardiometabolic Disease. N Engl J Med. 2014; 371:1131‑1141.
24. International Diabetes Federation. Diabetes Atlas. 2016. Seventh edition. www.diabetesatlas.org.
25. Kahn SE, 1, Hull RL, Utzschneider KM. Mechanisms linking obesity to insulin resistance and type 2 diabetes. Nature 2006;444, 840-846.
26. Steven S, Hollingsworth KG, Al-Mrabeh A, et al. Very-Low-Calorie Diet and 6 Months of Weight Stability in Type 2 Diabetes: Pathophysiologic Changes in Responders and Nonresponders. Diabetes Care. 2016 Mar 21. pii: dc151942.
27. Tilman D, Clark M. Global diets link environmental sustainability and human health. Nature. 2014;515, 518B522.
28. Hu, F. B. Globalization of diabetes: the role of diet, lifestyle, and genes. Diabetes Care. 2011; 34:1249B1257.
29. Shoelson SE, Lee J, Goldfine AB. Inflammation and insulin resistance. J Clin Invest. 2006;116 :1793B1801.
30. Shulman G. Ectopic Fat in Insulin Resistance, Dyslipidemia, and Cardiometabolic Disease. N Engl J Med. 2014; 371:1131‑1141.
31. International Diabetes Federation. Diabetes Atlas. 2016. Seventh edition. www.diabetesatlas.org.
32. Kahn SE, 1, Hull RL, Utzschneider KM. Mechanisms linking obesity to insulin resistance and type 2 diabetes. Nature 2006;444, 840-846.
33. Ali M. The Principles and Practice of Integrative Medicine Volume X: Darwin, Oxygen Homeostasis, and  Oxystatic Therapies.  3 rd. Edi. (2009) New York. Institute of Integrative Medicine Press.
34. Ali M. The Principles and Practice of Integrative Medicine Volume  XI: Darwin, Dysox, and Disease. 2000. 3rd. Edi. 2008. New York.  (2009) Institute of Integrative Medicine Press.
35. Ali M. The Principles and Practice of Integrative Medicine Volume  XII: Darwin, Dysox, and Integrative Protocols. New York (2009). Institute of Integrative Medicine Press.
36. Ali M. Oxygen, Inflammation, and Castor-Cise Liver Detox. Hormones. Townsend Letter-The examiner of Alternative Medicine. 2007. Published online. http://www.townsendletter.com/Dec2007/oxygen1207.htm
37. Ali  M. Philosophy and Science of holism in healing. APPNA Journal. 2015.
38. Ali M. Hyperinsulinism Associated With Breast and Prostate Cancer. Townsend Letter-The Examiner of Alternative Medicine. 2017;402:91-96.
39. Kamada N, Seo S-U, Zhiming C, et al. Role of the gut microbiota in immunity and inflammatory disease. Nature Reviews Immunology. 2013;12:321-335.
40. Grocott M, Richardson A, Montgomery H, et a. Caudwell Xtreme Everest: a field study of human adaptation to hypoxia. Critical care 2007;11:151.
41. Bahi-Buisson N, Roze E, Dionisi C, et al. Neurological aspects of hyperinsulinism-hyperammonaemia syndrome. Dev Med Child Neurol. 2008;50:945-9.
42. Stanley SA, Kelly L, Kaasmashri N, et al. Bidirectional electromagnetic control of the hypothalamus regulates feeding and metabolism. Nature. 2016  531:647–650.
43. Murphy KG, Bloom SR. Gut hormones and the regulation of energy bhomeostasis. Nature. 2006;444:854-859.

 

Link to Am Important Article

Shifting Focus From Glycemic Status to Insulin Homeostasis for Stemming Global Tides of Hyperinsulinism and Type 2 Diabetes

by
Majid Ali, MD, FRCS (Eng), FACP; Alfred O. Fayemi, MD, MSc (Path), FCAP; Omar Ali, MD, FACC; Sabitha Dasoju, MB, BS; Daawar Chaudhary; Sophia Hameedi; Jai Amin; Kadin Ali; Benjamin Svoboda

http://www.townsendletter.com/Jan2017/insulin0117.html

 


Can You Reverse Diabetes?

Majid Ali, M.D.

Are You Willing and Able to Try? Is So, Continue to Read.


 

Only You Can Answer the Question in the Title.
The information given below can help you. 


Dr. Ali’s Breakfast Shakes

Majid Ali, M.D/ Dr. Ali’s Breakfast Shakes Are Ideas, Not Products   Shakes for Weight Loss and Diabetes Reversal And Related Insulin-Smart Omelettes and Other Insulin-Smart Breakfasts for Insulin-Smart eating There is n…
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The Insulin Diet

Two Insulin Diet Plans Majid Ali, M.D. Insulin is the hunger hormone. This scientific simplicity forms the core of my Insulin Diet. I  prescribe it for my patients in one of its two forms: 1.             Insulin Diet One…

Insulin-Monitored Diabetes Reversal

Majid Ali, M.D.   Yes, almond butter is an insulin-smart food. Almond butter does not cause a blood sugar spike. So it does not cause blood insulin spike.   Almond butter is a good food also because it is rich in mineral…

The LOPI Way to Reversing Diabetes

Majid Ali, M.D. The Love-Oxygen-Prayer-Insulin (LOPI) Way for Reversing Diabetes Please consider this Path Away From Diabetes for yourself and for those you love. It is not a path of products. I is the path to truth, lov…

Insulin Toxicity of the Unborn

Majid Ali, M.D. The incidence of pregnancy-associated insulin resistance is rising worldwide, I think it is appropriately designated as insulin toxicity of the unborn. The incidence of pregnancy-associated insulin resist…

Optimal and Inappropriate Laboratory Testing For Assessing Insulin Homeostasis

Majid Ali, M.D. Grievous Errors in Insulin Testing   What Is Optimal Laboratory Insulin Testing? What Are Commonly Made Grievous Insulun Testing Errors?  Optimal laboratory testing for assessing insulin homeostasis is to…

Weight Loss – Truths and Mistruths

  Majid Ali, M.D. Another Hormone for Weight Loss and for Not Looking Like a Pear The only honest way of weight loss without losing health is eating less. The scientific truths behind this statement are: Insulin is the f…

Hyperinsulinism Associated With Breast and Prostate Cancer

Majid Ali, M.D. Published in the Journal Townsend Letter (2017;409:66-69 (August 2017)   Hyperinsulinism fans the fire of cancer. In this article, I present case studies to show diet and integrative therapies can restore…

Dr. Ali’s Breakfast Shakes

Majid Ali, M.D/

Dr. Ali’s Breakfast Shakes Are Ideas, Not Products


 

Shakes for Weight Loss and Diabetes Reversal


And Related Insulin-Smart Omelettes and Other Insulin-Smart Breakfasts for Insulin-Smart eating

There is never a valid reason for missing breakfast. So strong is my conviction on the subjects of the need and the optimal type of breakfast that I seldom complete a visit with one of my patients without addressing it. Indeed, next to the subject of chronic anger I devote more time energy—and energy— to the matter of “Dr. Ali’s breakfast” than any other subject. In this chapter, I present information about what I consider to be a good breakfast and offer my reasons for my position on the subject.

Many patients tell me they missed their breakfast because they were not hungry. I explain that was so because their metabolic rhythm has been disrupted. Rising at 7 am following a dinner at 8 pm the evening before, of course, means a fasting of 11 hours. Fasting means hypoglycemia and acidosis. Extending that period for another two or three or more hours essentially sets a person up for major hypoglycemic-hyperglycemic shifts that trigger insulin and adrenergic roller coasters. In individuals with neurotransmitter volatility — persons with predisposition for anxiety, sadness, or depression — extension of fasting can trigger any or all of those symptoms. For others without such vulnerability, it is really a matter of time until they also succumb to undue tiredness or mood difficulties caused by glucose-insulin-adrenaline-neurotransmitter shifts.

During the mid-1960s, as a house surgeon in Pakistan and later in England, I seldom ate a breakfast. I am not sure why that was so. Perhaps it was a macho thing — the real surgeons had to begin their mornings with something better than merely engaging Pakistani Praathas (heavily oiled and salted fried pita bread) or English porridge. More often than not, lunch was missed as well. By noon, I was nearly always in the midst of struggle with excising tissues soaked with bloody fluids. Sometimes there was a quick cup of coffee during the midday hours. At other times, I devoured a doughnut or a piece of pastry in the early afternoon hours. Now when I recall those days, I shudder to think who might have paid what price for the lack of any sense of nutrition — appalling ignorance, to be precise — of a driven, hypoglycemic, and inexperienced surgeon.

Dr. Ali’s Start-Low-Build-Slow Principle
In all guidelines for natural remedies for preserving health and reversing chronic diseases, I strongly advise readers to follow my Start-Low-Build-Slow Principle. Simply stated, this principle requires that an individual, in trying natural remedies, be cautious and closely observe how the body responds to natural remedies, beginning with small amounts or portions (as low as one-tenth the value on the first day, and doubling them every day until the recommended amount is reached.) If any negative senses are experienced, the item should be discontinued or taken in smaller amounts for longer periods of time to increase tolerance. It is important that a doctor be consulted to rule out the presence of serious coexisting or underlying conditions.

GOALS AND OBJECTIVES OF A GOOD BREAKFAST

A wholesome breakfast — in my view — should serve the following goals:

1. It should set the stage for an active, vigorous, and healthful day with sustained energy.
2. It should support the functions of the bowel, liver, hormone organs, brain, and other tissue.
3. It should sustain the long-term goals of healthful aging and preventing degeneratory disorders, such as heart disease, stroke, diabetes, Alzheimer’s disease, osteoporosis, and others.

With those goals in mind, the following objectives of a breakfast seem desirable to me:

1. Overhydration of cells throughout the body during the morning hours;
2. Maintenance of the blood glucose levels within a narrow healthful zone to avoid rapid hypoglycemic- hyperglycemic shifts, and to prevent insulin-adrenergic roller coasters initiated by those shifts;
3. Tonification of the bowel musculature and stimulation of the emptying reflexes;
4. Provision of special support for the hepatic redox-restorative detoxification pathways;
5. Provision of restorative oils for optimal biomembrane functions, especially in the brain;
6. Provision of raw material for structural and functional proteins, especially for generating oxystatic enzymes; and
7. Avoidance of toxic trans fatty acids, which are common in the standard American breakfasts.

Below, I describe my own breakfast, which seeks to meet the above goals and objectives of an optimal breakfast.

My Own Breakfast 3-4 Days a Week

I consider missing my breakfast a violation of the sacred temple that houses my spirit — an insult to my physical frame. I learned that crucial lesson the hard way. Earlier I referred to the absence of breakfast during my surgical days. That mistake was continued during two decades of my pathology work as well. My clinical experience and review of literature has convinced me that the single best:

1. That the premium nutrient for the brain is flaxseed oil;
2. That the single best nutrient for the liver is lecithin;
3. That the most desirable prescription for the integrity of bowel ecology is organic vegetable juice;
4. That the ideal formulation for the prevention of sugar-insulin-adrenaline roller coasters is a good protein powder, comprising 85 to 90% partially digested proteins; and
5. That the nutrient of choice for invigorating all the cells in the body is abundant water.

In view of those considerations, I prepare my own breakfast (within two to three minutes) on five or six days a week as follows:

1. I begin with a 28-ounce mug filled with spring water and one ounce of seltzer water (seltzer water is unnecessary for those who prefer plain water).
2. I drink the above-mentioned fluid volume in portions of five to seven ounces at intervals, doing limbic exercise — non-competitive exercise—between drinking. I take my probiotic protocol (Bifidobacterium andAcidophilus) when I begin to drink water.
3. I continue drinking that amount of water, taking additional supplements, until the mug is empty.
4. Next, I prepare a 30-ounce protein drink as shown in Table 1.
5. I begin drinking the above protein protocol, again consume five to seven ounces at a time, continuing my limbic exercise and taking additional supplements until both the complement of supplements and the protein formula is finished.
6. On weekends, my wife and I commonly take an egg breakfast with some fruit.

At the Institute, my colleagues and I use the following four P&P (partially digested protein) protocols: (1) #1 containing %90% mixture of proteins derived from eggs and milk; (2) # 2 containing 90% soy proteins; (3) # 3 containing 75 to 80 % rice protein; and (4) # 4 containing about 90% of whey protein. Proteins derived from other sources, such as vegetables, may be substituted for one or the other of the above choices. In Table 2, I make some recommendations for the choice of vegetables for preparing fresh juices:

The flaxseed oil in the protein drink may be replaced by one of the following oils: olive, safflower, sunflower, sesame, pumpkin, avocado, and almond. Additional comments on this subject are included in the next section.

I use the Very Veggie brand of bottled organic vegetable juice produced by Knudson Co. For those who can find the time, freshly squeezed vegetable juice is clearly preferable.

Table 1. Dr. Ali’s Breakfast
Five Days of the Weeks

Protein                                    Two heaping tablespoons

Flaxseed                                  Two heaping tablespoons

Lecithin (all natural)                   One heaping tablespoon

Vegetable juice, organic             15 ounces

Water                                     15 ounces

Insulin-Smart Omelettes and Other Insulin-Smart Breakfasts for Insulin-Smart eating

When we do what is right, habit makes it agreeable. That certainly is true of eating choices. Taste is an acquired faculty. Organic vegetable juice added to my protein drink now appeals to me much more than any fruit juice. It was not so when I began. (Fried brain is not a delicacy for me now as it once was in my childhood in Pakistan.) The habit one grows into for one’s breakfast is as much a part of the life’s track as any other. I explain those basic aspects of a wholesome breakfast to every patient who consults me.

Some patients readily follow my breakfast plan closely, begin to savor it soon, and report good results within weeks. Others take a slower approach, adopting my plan partially. Some patients initially find my prescription for breakfast unappetizing. The majority of them settle into them nicely weeks or months later. Yet others ask if they can replace organic vegetable juice with milk (cow’s, goats, rice or soy). Others wish to add one-half of a banana or peach or other types of fruit for enhanced taste. Except in patients with disturbing symptomatology related to rapid hypoglycemic-hyperglycemic shifts, I accept their modifications.

I do wholeheartedly endorse an egg breakfast. Eggs have been maligned for decades by practitioners of pharmacologic medicine on the grounds that eggs raise blood cholesterol levels. I dismiss that as non-sense coming from ill-informed individuals. Not a single study has shown that eggs increase the incidence of cardiovascular disease. Indeed, some reports suggest that eggs — an excellent source of high-quality liver-friendly lecithin —actually lower blood cholesterol levels.

Plain yogurt with freshly ground flaxseed makes for an excellent breakfast. Some fruit may be added to that for persons without immune disorders. Other good breakfast options are soy products commonly recommended by macrobiotic enthusiasts.

Oatmeal breakfast used to be a favorite of nutritionists of bygone eras. Nearly all nutritionists that I have discussed this subject with in recent years told me they now do not recommend oatmeal or any other starch breakfasts anymore. I believe that is because the abuse of antibiotics and massivesugar overload in the general public has so stressed the bowel ecosystem that even so-called healthy starches now carry the hazard of further feeding the sugar- eaters in the gut and so contribute to ongoing disruption of the bowel ecology.

In some cultures, fish, poultry, and various meats are consumed for breakfast. Those items are very desirable as sources of proteins. The important point here is that such meats should not be highly processed, nitrated, or otherwise contain high contents of oxidized fats.

Dr. Ali’s Recipesi for Breakfast and  Related Articles
* Dr. Ali’s Insulin-Wise Breakfast
* Dr. Ali’s Insulin-Wise Breakfast – Personalized

 

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