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Reversing Diabetes – Lesson Six

Majid Ali, M.D.

Short Cuts to Materials Cited in This Lesson

How Do You Reverse Diabetes?

A Short Video  – https://vimeo.com/96366665


How Different Meals Affect

Metabolism Differently?


The List of Relevant Links for This Lesson 


The Deeper One’s Understanding the

the Food-Insulin Relationships,

the Higher the Probability of Reversing One’s Diabetes.


The Case Study Below Shows Diabetes Appears With One Type of Meal And Disappears With Another. With Time, Diabetes Is Reversed With Good Meals and Does Not Return With Infrequent Exceptions.


Learning the Difference Between the Effects

of Carbohydrate and Non-Carbohydrate Meals


 

A Revealing Case Study

 A Profile With Sugar (Glucose) Load

Table 1. Insulin and Glucose Profiles After a 75-Gram Glucose Load Of a Response of

50-Yr-Old 5’ 11” Man Weighing 195 lbs. In Apparent Excellent Health. His Blood  A1c

 Value Is 6.6%. There Is No Past History of Diabetes.

7.15.2016

Fasting

½ Hr

1-Hr

2-Ht

3 Hr

Insulin uIU/mL

27.1

43.6

81.8

109.8

69.5

Glucose mg/mL

114

193

256

243

136

The A1c Test Done With the Blood Insulin and Glucose Tests Showed a Rise From 6.6% to 6.9%.

 


Questions Concerning Table 1

tudy

  1. What does A1c value of 6.6% mean?
  2. Does the blood insulin level 43.6 mean the study subject has hyperinsulinism (excess insulin)?
  3. Does the blood sugar level of 193 mean that the study subject has prediabetes?
  4. Does the 1-hr blood glucose value of 256 mean that the subject has Type 2 diabetes?
  5. Does the 2-hr blood glucose value of 243 mean that the subject has Type 2 diabetes?
  6. Does the 2-hr blood insulin level of 109.8 clearly establish the diagnosis of hyperinsulinis?
  7. Do the data in Table 1 clearly establish the diagnosis of Type 2 diabetes?
  8. What might be the treatment options for the man in the above case study as shown in Table 1?
  9. What is the primary goal in treating someone with Type 2 diabetes and very high insulin levels?

 

Insulin And Glucose Profiles After

A Protein + Fat Meal

Table 2. Insulin and Glucose Profiles After a 75-Gram Glucose Load Of a Response of

50-Yr-Old 5’ 11” Man Weighing 195 lbs. In Apparent Excellent Health. His Blood  A1c

 Value Is 6.6%. There Is No Past History of Diabetes.

7.15.2016

Fasting

½ Hr

1-Hr

2-Ht

3 Hr

Insulin uIU/mL

15.8

81.8

11

Glucose mg/mL

94

94

89

Can the diagnosis of Type 2 diabetes be established by the data in this table?

Can the diagnosis of hyperinsulinism be established by the data in this table?

  1. Do the data in Table 2 show that diabetes has been reversed?
  2. Do the data in Table 2 show that there is a very high probability that diabetes can be reversed if the treatment plan of insulin detox is continued?
  3. What is the meaning of the fasting insulin level of 27.1 in the initial test falling to 15.8 in the follow-up test?
  4. What is the meaning of the fasting blood glucose level of 114 in the initial test falling to 94 in the follow-up test?
  5. How would the study subject know if his diabetes has been reversed?
  6. If diabetes is reversed, can it return?
  7. Would it be more difficult to reverse diabetes if it were to return?

Information for Answers to the Questions 

  1. A1c test of 6.6% means that the subject has been running bloos sugar levels seen in Type 2 diabetes.
  2. Ideally, men should not have the peak insulin level in the profile higher than 25 uIU/mL.
  3. A 1-hr glucose level of above 200 mg/dL is generally considered diagnostic of prediabetes.
  4. A 2-hr and 3-hr glucose levels of above 200 mg/dL are considered diagnostic of Type 2 diabetes.
  5. The desirable treatment of newly diagnosed diabetes is first to try to reverse it with food choices in the kitchen and relevant natural remedies.
  6. The primary goal in treating someone with Type 2 diabetes and very high insulin levels is to: (1) make insulin Work; (2) lower blood sugar levels without drugs; and (3) reverse diabetes.
  1. A test done with non-carbohydrate meal challenge is the best way to assess how the metabolism of an

Individual with Type 2 diabetes responds to non-carbohydrate meal. It clearly shows the way to reverse diabetes.


                 Dr. Ali’s Video Series

https://vimeo.com/search?q=how+do+you+reverse+diabetes%20majid%20ali

Insulin-Toxic Obesity
Fats Are Not Fattening Americans
 Demand Nor Offer Forgiveness, Act Forgiveness

What Is the Most Important Question in Science, Health, and Healing
What Is the Second Most Important Question in Science, Health, and Healing

What Can Blood Cells Teach About Diabetes?

 


What Can A Lemon Teach About Diabetes?


Reversing Type 2 Diabetes Needs Learning, Eating the Right Foods, Insulin Detox, and patients.


 

How Do You Reverse Diabetes?

A Short Video  – https://vimeo.com/963

How Do You Reverse Diabetes

https://vimeo.com/963

 

 

Free Access Diabetes Library

Majid Ali, M.D.


Library of Articles and Videos

Dr. Ali’s Three-Part Diabetes Course
 
Dr. Ali’s Diabetes Course – Part 1: The Basics of Diabetes
https://alidiabetes.org/2016/06/27/dr-alis-diabetes…-part-one-basics/ ‎
 
Dr. Ali’s Diabetes Course – Part 2: Insulin Detox – Beyond Sugar Talk
https://alidiabetes.org/2016/07/11/dr-alis-diabetes-course-part-two-2/ ‎
 
Dr. Ali’s Diabetes Course – Part 3:
https://alidiabetes.org/2016/07/25/dr-alis-3-part-d…ourse-part-three/
 
 
Breakfasts 2016
 
MMM
 
Lab Ref Ranges
 
 
 
Reversing Diabetes Pack
Reversing Diabetes – Lesson One
DR. ALI’S 3-PART DIABETES COURSE PART TWO
DR. ALI’S 3-PART DIABETES COURSE – PART THREE
Reversing Diabetes – Lesson Four
https://alidiabetes.org/2016/08/15/reversing-diabetes-lesson-four/
Reversing Diabetes – Lesson Five
Reversing Diabetes – Lesson Six
Reversing Diabetes – Lesson Seven Spiritual Speak
Reversing Diabetes – Seven Simple Lessons
Diabetes Recipes
DR. ALI’S 3-PART DIABETES COURSE – PART THREE
DR. ALI’S 3-PART DIABETES COURSE PART TWO
alink: https://alidiabetes.org/2016/08/15/reversing-diabetes-lesson-four/ ‎Edit Get

DIABETES VIDEO LIBRARY

Diabetes videos part 1 | The Ali Academy Community

In this 55-minute video seminar, Professor Majid Ali, M.D. discusses the causes, clinical features, and consequences of insulin toxicity, including pre-diabetes …

Diabetes Insulin Videos – Ali Healing Community

Majid Ali, M.D. Links to Videos on Prevent and Reverse Diabetes What is Diabeteshttps://www.youtube.com/watch?v=vTUFY2It-vQ What Is Insulin? What Are Its …

Majid Ali, M.D. * Insulin Toxicity De-mystifies the Metabolic Syndrome …

Jun 28, 2012 – Uploaded by majid ali

The true mature of the metabolic syndrome is insulin toxicity. The term metabolic syndrome creates creates …

Majid Ali, M.D. * Can You Increase Natural Insulin in Diabetes …

Jun 5, 2012 – Uploaded by majid ali

The answer is YES in many cases. I illustrate this with a case study. In advanced stages of diabetes Type 2 …

Majid Ali MD, Castor Oil Rubs for Insulin Detox for Weight Loss and …

https://vimeo.com › Majid Ali › Videos
Jan 24, 2015 – Uploaded by Majid Ali

Type 2 diabetes is an insulin-toxicity state for years before the body reserves ofinsulin are depleted and the …

Majid Ali MD, Dr. Ali’s Book on Reversing Diabetes – Dr. Ali’s Plan for …

https://vimeo.com › Majid Ali › Videos
Nov 6, 2014 – Uploaded by Majid Ali

I outline the contents of this book on reversing diabetes Type 2. I explain how it begins with insulin toxicity …

Insulin spikes | Ali Diabetes

Posts about Insulin spikes written by Majid Ali MD. … Reversing Prediabetes and Diabetes With 3D Plan: Insulin-Wise and Insulin-Unwise Foods and Meals. Posted on July 24, 2017 by Majid … Video for majid ali diabetes insulin videos ▷ 5:57.

Insulin-Monitored Diabetes Reversal | Ali Diabetes

Sep 30, 2017 – Majid Ali, M.D. Yes, almond butter is an insulin-smart food. … List of Videos for Learning and Implementing Dr. Ali’s Insulin-Based Diabetes …

3D Insulin Protocol | Ali Diabetes

Posts about 3D Insulin Protocol written by Majid Ali MD. … Diabetes is a two-faced disease, one withinsulin toxicity and the other with insulin depletion: this diabetes duality in itself is most revealing. ….. https://vimeo.comMajid AliVideos.

Dr. Ali’s Insulin Reduction Protocol

Majid Ali, M.D. … For individuals with pre-diabetes with insulin toxicity but without high blood sugar levels, my … I present this subject at length in my book entitled “Dr. Ali’s Plan for Reversing Diabetes” and in a 40-minute video seminar that can …

Shortlink

Dr. Ali’s Breakfast Shakes

 

Restoring Insulin Homeostasis, Reversing Diabetes

Majid Ali, M.D.

The Work of True Physicians Does Not Belong to Them, just As Their Words Do Not Belong to Them.  

The Healing of True Physician’s Belongs to Their Patients, ,Just As the Words of True Writers Belong to Their Readers.


First Things First

I.  There are two true markers of real enduring health:

                                                         1. Oxygen health

                                                         2.  Insulin health

II. To understand health is to understand oxygen health and insulin health.  

III. To understand disease is to understand inflammation.

IV. No healing is possible without physiological healing.

V. No disease is possible without pathologic inflammation.

VI. Pathologic inflammation results from disrupted oxygen and insulin signaling.


Insulin Health

Dr. Ali’s Diabetes Library 

Dr. Ali’s Diabetes Course – Part 1: The Basics of Diabetes
https://alidiabetes.org/2016/06/27/dr-alis-diabetes…-part-one-basics/ ‎
 
Dr. Ali’s Diabetes Course – Part 2: Insulin Detox – Beyond Sugar Talk
https://alidiabetes.org/2016/07/11/dr-alis-diabetes-course-part-two-2/ ‎
 
Dr. Ali’s Diabetes Course – Part 3:
https://alidiabetes.org/2016/07/25/dr-alis-3-part-d…ourse-part-three/ 
 
Diabetes Recipes
 Reversing Diabetes – Lesson One
 Reversing Diabetes – Lesson Two
 Reversing Diabetes – Lesson Three
Reversing Diabetes – Lesson Four
Reversing Diabetes – Lesson Five
Reversing Diabetes – Lesson Six
 Diabetes Recipes

Dr. Ali’s Insulin Library

Spiritual Healing Course byMajid Ali, M.D.
 What Is Insulin? What Are Its Functions?
 
 Insulin Detox for Wight Loss and Diabetes Reversal
 
 I’m Hungery After Meals. Why?
 Insulin Buddy and Fatty Liver
 
 What is the Evidence That Neuropathy Is Caused by Insulin Toxicity?
 
Obesity Is Cellular Inflammation
 
Dr. Ali’s Best Anti-Insulin Toxicity Breakfast
 
 Gestational Diabetes Is Insulin Toxicity of the Unborn – Part Two
Your Child – Hyperactive or Hypoglycemic?
 
Why Do I Consider Blood Insulin Test to be the Most Important Test for Metabolism and Diabetes 
Insulin-Toxic Obesity
What Is the Most Important Question in Science, Health, and Healing
 
What Is the Second Most Important Question in Science, Health, and Healing
 
Insulin Videos
 
 
What is Diabetes
Insulin Toxicity De-mystifies Syndrome X
Don’t Trust A1c for Diabetes Diagnosis, Please!
 
 Recipes for Insulin Toxicity – Majid Ali, MD
 
 
Almond Butter Snack for Losing Weight and Reversing Diabetes, An Excellent Choice
 
 
Peanut Butter or Hemp Seed Butter Peanut Butter Snack for Weight Loss and Diabetes Reversal
  
 
 Dr. Ali’s Insulin Course, Basics
 
What Is the Problem With Calorie Counting?
Is a Calorie a Calorie a Calorie?
 
 
Is Insulin Excess Bad for the Heart?
 
 
 
What Is Diabetes? Majid Ali, M.D. With Ben Svoboda
 
Is Excess Insulin Toxic to Nerves
 
 
 
 
Majid Ali, M.D. – Is a Calorie a Calorie a Calorie?
 
I’m Hungry After Meals. Why?
 
 
Recipes for Insulin Toxicity – Majid Ali, MD
 
 
 
Almond Butter Snack for Losing Weight and Reversing Diabetes, An Excellent Choice
 
 
Peanut Butter or Hemp Seed Butter Peanut Butter Snack for Weight Loss and Diabetes Reversal
 
 
 
 Diabetes and insulin Majid Ali MD
Why Do I Consider Blood Insulin Test to be the Single Most Important Test
 
What Is Your Child’s Peak Insulin Level? Is She or He Insulin-Toxic?
 

Reversing Diabetes Lesson Seven – Diabetes, Inflammations, and Infections

 

Majid Ali, M.D.

My Three Top Priorities for Patients With Inflammatory Disorders: 

First, Think of Coexisting Infections.

Second, Think of Cellular Energy and Insulin Status.

Third, Be Holistic and Integrative in the Science and Philosophy of Caring.


Two Core Messages

First, all forms of inflammations, with or without infections, increase the risk of diabetes, and diabetes increases the risk of all  types of inflammations and infections.

Second, for acute infections, microbes require first attention. For chronic infections, one’s own human cells require first attention.


Two Essentials

  1. All Inflammations and Infections begin with fermentation in the bowel and in the mind.

  2. Prevention and control of inflammation and infection must begin with control of fermentation in the gut and the mind.


A Treasured Lesson From My Patients:

Knowing Oxygen and Insulin for Health.

Knowing Inflammation For Healing.


Three Questions

Is inflammations always bad?

What weakens immunity to inflammations and infections in diabetes?

What causes insulin toxicity (hyperinsulinism)?


Think Seven

for Recurrent and Chronic Infections in Diabetes

  1. Think Prayer

  2. Think Oxygen

  3. Think Insulin

  4. Think Bowel

  5. Think Nutrients, Spices and Herbs

  6. Think Anti-inflammatory foods

  7. Think Specific Remedies 

Think Prayer

Pray, Just pray –  For whatever, whomever, wherever

Think Oxygen

Breathe slow, just breathe out slow – for however, however  long,  wherever

Think Insulin

Pray, Just pray –  For whatever, whomever, wherever

Think Bowel

Breathe slow, just breathe out slow – whereever, for however long.

Think Nutrient, Spices, Herbs

Begin low, build slow, be a scientist, try it, observe the effects, and move on.

Anti-Inflammatory Foods

Foods that cause sugar spikes which trigger insulin spikes are proinflammatory foods. Non- toxic foods that do not cause sugar spikes are anti-inflammatory foods.

Specific Remedies

Chromium, vanadyl, neem leaves, and others.



My First Encounter With the Diabetes-Infection Axis

One of my sharpest recollection of events in a medical clinic in 1961 is of how a professor diagnosed diabetes by looking at the neck of a new patient. The patients had a carbuncle of the neck which is considered diagnostic of diabetes because the disease does not allow to let this skin infection heal. That experience returns whenever I think of the diabetes-infection axis.


Four Historical Footnotes

1876 .          W. Epstein controls sugar leak in diabetes with aspirin.

                      Berliner Klinicsche 1876;3:337-340.

1901.           R. Williamson control sugar in urine in diabetes with aspirin. 

                      British Medical Journal. 1901;1:760-762.

1957.            Reid  J. Macdougall AL Stop insulin injections with aspirin.

                     British Medical Journal. 1957;8:418-428.   

1921.          Insulin discovered by the Canadian physician Frederick Banting

                    and Student Charles H. Best.


Types of Infections

  1. Acute .   Viral, Bacterial, yeast, parasitic

  2.  Subacute .  Viral, Bacterial, yeast, parasitic

  3.  Chronic .  Viral, Bacterial, yeast, parasitic

Two important microbial species causing infections in diabetes ate Staph aureus and Candida species.


The Gut-Diabetes Connections

 

  • Throat

  • Esophagus

  • Stomach

  • Small intestine

  • Large intestine


Molecular Mechanisms of the Diabetes-Infections Axis

1.  Less circulation, less oxygen

2. Reduced number and efficiency of hunter-immune cell function                              (decreased phagocytic function)

3. Reduced number and functional deficits of immune white blood cells

4. Sluggish movements of immune cells

5. Increased apoptosis (planned cell suicide)

6.  Low levels of inflammatory cytokines

7. Compromised complement system defense molecules

8.  Low concentrations and efficiency of antibodies


Acute Infections and Chronic Infections

Preferred Natural Remedies

  1. Optimal hydration

  2. Dr. Ali’s Spicy Smoothie

  3. Probiotics (yogurt, Kiefer, Acidophillus)

  4. Turmeric and Vitamin C (1000 mg of each) four times a day

  5. Oregano oil

  6. Special Nutrients (Zinc lozenges, Magnesium, Potassium, Taurine)

  7. Antibiotics, If really needed

  8. Antifungal Spices, Herbs, and Medications. 

One of my sharpest recollection is of a patient that I saw as a student in 1961. He came to a medical clinic with a carbuncle (deep seated chronic skin infection) on the back of the neck. My professor took one look at the carbuncle and clinically diagnosed diabetes,  which proved right on blood tests. It would be 54 years before I learned the about the following three publications clearly establishing the insulin-inflammation connections.


Three Neglected Insulin Lessons From the Past.

1876 .          W. Epstein controls sugar leak in diabetes with aspirin.

                      Berliner Klinicsche 1876;3:337-340.

  1.           R. Williamson control sugar in urine in diabetes with aspirin. 

                      British Medical Journal. 1901;1:760-762.

  1.            Reid  J. Macdougall AL Stop insulin injections with aspirin.

                     British Medical Journal. 1957;8:418-428.   

 I anticipate three questions here: (1) the above three reports refer to Type 2 diabetes (T2D), why do is call them “three insulin lessons”? (2) what lights do the insulin-inflammation connections shed on neurodevelopmental biology? and (3) specifically, what clinical imperatives might be recognized to restore neuronal progenitor cell progression to mature “speech neurons, as well as other neurons involved involved with the autism spectrum, dysautonomia, and related neurodevelopemental disorders? Here are brief answers: (1) hyperinulinism predates T2D in all cases and maternal hyperinsulinism is a hazard for neuronal progenitor cell progression of the unborn child; (2) unrecognized hyperinsulinism in ASD and CID  during prenatal to threatens poses serious threats to developmental biology.


A Crucial Question

Why did I dig them out for investigations into the molecular basis of neurodevelopmental  studies discussing the results o I bring The answers: (1) unrecognized maternal hyperinsulinism is a hazard for neurobioilogy of the unborn baby; (2) unrecognized hyperinsulinism in early postnatal life is a hazard for progenitor cell progression; and (3)  for neurobioilogy of the unborn baby rain; T2D, is first and foremost, an insulin toxicity problem. Hyperinsulinism predates T2D. This relationship has been recognized for decades. The author has never seen T2D develop without hyperinsulinism preceding it. I have published many case studies toType 2 to illustrate these observayions. 

Proinflammatory Cytokines Induced Insulin Resistance

In 1993, 117 years after the first published report of antidiabetes effects of aspirin, the  effects of aspirin,  the proinflammatory cytokine NFK-a  was shown to induce insulin resistance.25,27 This seminal advance led to rapid recognition of similar anti-insulin, pro-diabetes effects of other inflammatory cytokines,  adipokines (leptin, adiponectin, and others produced by fat cells), resistin, visfatin, PAI-1, IL-6, angiotensinogen,  retinol-binding protein-4, serum amyloid A (SAA), and others.28-33  


Insulin Adjudicates Physiological and Pathological Inflammation


The Gut-Diabetes Connection

 

  • Throat

  • Esophagus

  • Stomach

  • Small intestine

  • Large intestine

Digestion starts within the mouth by the action of the enzymes in saliva. It then takes full effect within the stomach and some nutrients are also absorbed into the bloodstream here. Partially digested food known as chyme then undergoes further digestion mainly in the first part of the small intestine known as the duodenum. The small intestine, or small bowel, is the longest part of the gut and gradually the food is completely digested and almost all the nutrients are absorbed into the bloodstream.


Important Sites of Diabetes-Related Infections

  1.  Infections of the Urinary Tract

  2.  Vaginal Tract

  3. Skin

  4. Lungs and Pulmonary Tract


 Molecular Mechanisms of the Diabetes-Infections Axis

1.  Less circulation, less oxygen

2. Reduced number and efficiency of hunter-immune cell function                              (decreased phagocytic function)

3. Reduced number and functional deficits of immune white blood cells

4. Sluggish movements of immune cells

5. Increased apoptosis (planned cell suicide)

6.  Low levels of inflammatory cytokines

7. Compromised complement system defense molecules

8.  Low concentrations and efficiency of antibodies


Decreased mobilization of polymorphonuclear leukocytes, chemotaxis, and phagocytic activity may occur during hyperglycemia.[4,9,10] The hyperglycemic environment also blocks the antimicrobial function by inhibiting glucose-6-phosphate dehydrogenase (G6PD), increasing apoptosis of polymorphonuclear leukocytes, and reducing polymorphonuclear leukocyte transmigration through the endothelium.[4] In tWeak 

s

4.  Complement

Complement

The complement system is one of the main mechanisms responsible for the humoral immunity. It consists of serum and surface proteins whose main functions are to promote the opsonization and phagocytosis of microorganisms through macrophages and neutrophils and to induce the lysis of these microorganisms. Moreover, complement activation products provide the second signal for B-lymphocyte activation and antibody production.

Although some studies have detected a deficiency of the C4 component in DM,[5,6] this reduction of C4 is probably associated with polymorphonuclear dysfunction and reduced cytokine response.[2,5]

Inflammatory cytokines

Mononuclear cells and monocytes of persons with DM secrete less interleukin-1 (IL-1) and IL-6 in response to stimulation by lipopolysaccharides.[2,4] It appears that the low production of interleukins is a consequence of an intrinsic defect in the cells of individuals with DM.[2,7] However, other studies reported that the increased glycation could inhibit the production of IL-10 by myeloid cells, as well as that of interferon gamma (IFN-γ) and tumor necrosis factor (TNF)-α by T cells. Glycation would also reduce the expression of class I major histocompatibility complex (MHC) on the surface of myeloid cells, impairing cell immunity.[8]

Polymorphonuclear and mononuclear leukocytes

Decreased mobilization of polymorphonuclear leukocytes, chemotaxis, and phagocytic activity may occur during hyperglycemia.[4,9,10] The hyperglycemic environment also blocks the antimicrobial function by inhibiting glucose-6-phosphate dehydrogenase (G6PD), increasing apoptosis of polymorphonuclear leukocytes, and reducing polymorphonuclear leukocyte transmigration through the endothelium.[4] In tissues that do not need insulin for glucose transport, the hyperglycemic environment increases intracellular glucose levels, which are then metabolized, using NADPH as a cofactor. The decrease in the levels of NADPH prevents the regeneration of molecules that play a key role in antioxidant mechanisms of the cell, thereby increasing the susceptibility to oxidative stress.

Regarding the mononuclear lymphocytes, some studies had demonstrated that when the glycated hemoglobin (HbA1c) is <8.0%, the proliferative function of CD4 T lymphocytes and their response to antigens is not impaired.[4]

Antibodies

Glycation of immunoglobulin occurs in patients with diabetes in proportion with the increase in HbA1c, and this may harm the biological function of the antibodies.[4] However, the clinical relevance of these observations is not clear, since the response of antibodies after vaccination and to common infections is adequate in persons with DM.[4]


How Do You Reverse Diabetes Majid Ali MD on Vimeo


Reversing Diabetes – Lesson One

https://alidiabetes.org/?s=reversing+diabetes+-+lesson+-+one

Reversing Diabetes – Lesson Two

https://alidiabetes.org/?s=reversing+diabetes+-+lesson+-+two+

Reversing Diabetes – Lesson Three

https://alidiabetes.org/?s=reversing+diabetes+-+lesson+-+three


Reversing Diabetes – Lesson Four

https://alidiabetes.org/?s=reversing+diabetes+-+lesson+-+four

Reversing Diabetes – Lesson Five

https://alidiabetes.org/?s=reversing+diabetes+-+lesson+-+five

Reversing Diabetes – Lesson Six

https://alidiabetes.org/?s=reversing+diabetes+-+lesson+-+six


DR. ALI’S DIABETES COURSE – OUTLINE

Majid Ali, M.D.

A Special Note: I suggest that individuals considering this FREE course scan this outline at least twice  the to become familiar with the organization of its three parts, and then decide how to pace themeselves. Search box of this web site will readily take them to the subjects and articles of their interest.



Three Parts of Dr. Ali’s Diabetes Course

Part 1:

The Basics of Diabetes — causes, symptom-complexes, clinical course, treatment options

Part 2:

Shift from glycemic status to insulin homeostasis (Getting away from the “sugar talk.” Knowing the truth.)

Part 3:

Clearing insulin toxicity, preventing diabetes, reversing Type 2 Diabetes, preventing and/or controlling complication, preparing to teach the course to otheres.


Limits on Students Taking the Course

  1. There are no limits who should take this FREE course..
  2. No medical background is necessary.
  3. The Course can be completed in six weeks or six months, or on an individual’s own pace.

 

Course Study Partners

Studying anything with a study partner is always an excellent idea. It especially valuable for this course whether taken for weight loss or diabetes concerns (family history, recent or old diagnosis, recent diagnosis of diabetes-related fatty liver, kidney failure with threat of dialysis, vision problems, cardiovascular disorders, or worries about memory loss and brain shrinking.


Course Study  Textbook

Dr. Ali’s Diabetes Reversal Plan

Dr. Ali’s Insulin Toxicity Workbook


 

Basic Questions

  1. Is diabetes a sugar problem? No.
  2. Is diabetes an insulin  toxicity problem? Yes.
  3. Can diabetes be reversed? Yes. Nearly in all cases of Type 2 diabetes in the first three to fives years of diagnosis.
  4. The above answers are unexpected for me. Why is it so?  (Please answer this question yourself.)  

 

Diabetes can neither be diagnosed early nor reversed if it is seen as a sugar problem.


 

Body Organs of Special Interest inOxygen Models of Diabetes and Insulin Toxicity

  1. Gut
  2. Liver
  3. Thalamus in the Brain
  4. Muscles
  5. Pancreas

Why Is the pancreas gland that produces insulin so low in the order of body organs?

I invite readers to keep this question in the mind as they consider Dr. Ali’s Course on Diabetes Reversal.


Scientific Basis of Insulin-Based Diabetes Reversal

Dr. Ali’s Diabetes Course is based on the sciences of: 

  1. Molecular Biology of Oxygen
  2. Insulin homeostasis

Study of insulin toxicity defines the problem. Solutions are in the study of oxygen.


Two Dimensions of Dr. Ali’s Diabetes Course for Reversing Type 2 Diabetes

          ☞ Insulin Toxicity  (to Know the Problem)

         ☞ Diabetes Reversal (to Know the Solution)


Five Threats to Humankind:

  1. Developmental Challenges of the Unborn
  2. Diabetes
  3. Dialysis
  4. Dementia
  5. Disability

All  five are rooted in insulin toxicity. I anticipate that some readers will roll their eyes on the first item listed above. That only means they are not aware of the frequency with which hyperinsulinism is encountered in children with autism, dysautonomia, OCD, POTS, and related neurodevelopmental challenges faced by children in prenatal and postnatal lives.


 

Oxygen Models of Insulin Toxicity and Diabetes Reversal Five Threats to Humankind:

Dr. Ali’s Insulin Toxicity Course and Dr. Ali’s Diabetes Reversal Course are based on Oxygen Models of Diabetes and Insulin Toxicity. Simply stated, these models explains all aspects of Type 2 diabetes—causes, clinical course, consequences, and control—on the basis of disturbed oxygen function. A full description of these models is included at the end of this article.


Body Organs of Special Interest inOxygen Models of Diabetes and Insulin Toxicity

  1. Gut
  2. Liver
  3. Thalamus in the Brain
  4. Muscles
  5. Pancreas

Why Is the pancreas gland that produces insulin so low in the order of body organs?

I invite readers to keep this question in the mind as they consider my Course on Diabetes?


 

The Gut-Diabetes Connection

 The Gut-Diabetes Connections

  • Throat
  • Esophagus
  • Stomach
  • Small intestine
  • Large intestine

Digestion starts within the mouth by the action of the enzymes in saliva. It then takes full effect within the stomach and some nutrients are also absorbed into the bloodstream here. Partially digested food known as chyme then undergoes further digestion mainly in the first part of the small intestine known as the duodenum. The small intestine, or small bowel, is the longest part of the gut and gradually the food is completely digested and almost all the nutrients are absorbed into the bloodstream.


The Thalamus-Feeding-Weight-Diabetes Connections

Picture

The Quick Facts

Location: Part of the forebrain, below the corpus callosum
Function: Responsible for relaying information from the sensory receptors to proper areas of the brain where it can be processed
The thalamus in the brain has special centers for glucose and regulates some aspects of  sensory information that is being transmitted to the brain.
 

 

What Is More Important in Diabetes?

In Beta Cells of the pancreas where insulin is produced?

Or in cell membranes where it moves receptor proteins?

muscleWhere Insulin Is Produced Or Where It Is Used?


What Is Insulin? Where Does It Come From?

The pancreas is a long, slender organ, most of which is located posterior to the bottom half of the stomach.  Although it is primarily an exocrine gland, secreting a variety of digestive enzymes, the pancreas has an endocrine function. Its pancreatic islets—clusters of cells formerly known as the islets of Langerhans—secrete the hormones glucagon, insulin, somatostatin, and pancreatic polypeptide (PP).

Pancreas

This diagram shows the anatomy of the pancreas. The left, larger side of the pancreas is seated within the curve of the duodenum of the small intestine. The smaller, rightmost tip of the pancreas is located near the spleen. The splenic artery is seen travelling to the spleen, however, it has several branches connecting to the pancreas. An interior view of the pancreas shows that the pancreatic duct is a large tube running through the center of the pancreas. It branches throughout its length in to several horseshoe- shaped pockets of acinar cells. These cells secrete digestive enzymes, which travel down the bile duct and into the small intestine. There are also small pancreatic islets scattered throughout the pancreas. The pancreatic islets secrete the pancreatic hormones insulin and glucagon into the splenic artery. An inset micrograph shows that the pancreatic islets are small discs of tissue consisting of a thin, outer ring called the exocrine acinus, a thicker, inner ring of beta cells and a central circle of alpha cells.

The pancreatic exocrine function involves the acinar cells secreting digestive enzymes that are transported into the small intestine by the pancreatic duct. Its endocrine function involves the secretion of insulin (produced by beta cells) and glucagon 

Two Dimensions of Dr. Ali’s Diabetes Course for Reversing Type 2 Diabetes

          ☞ Insulin Toxicity Course (to Know the Problem Well

         ☞ Diabetes Reversal Course to Know then Solution Well for Reversing Diabetes?

 

I offer my course in two parts: (1) Dr. Ali’s insulin Toxicity Course; and (2) Dr. Ali’s Diabetes Reversal Course. I attribute the two parts of this course to myself for the simple reason that it makes it easier for people to find it on the internet.

Dr. Ali’s Diabetes Course and Insulin Toxicity Courses are free for everyone, and are posted athttp://www.alidiabetes.org. For my free recipes, please go to http://www.alidiabetes.org. 

Should anyone or any institution wish to teach this course, please send me a note and I will send you written permission to do so without any cost.


Dt. Ali’s Basic, Intermediate, and Advanced Diabetes Courses

 My both Diabetes Course and Insulin Toxicity Course are subdivided into three levels as follows:

  1. Ali’s Basic Diabetes Course
  2. Ali’s Intermediate Diabetes Course
  3. Ali’s Advanced Diabetes Course
  4. Ali’s Basic Insulin Toxicity Course
  5. Ali’s Intermediate Insulin Toxicity Course
  6. Ali’s Advanced Insulin Toxicity Course

What Does the Basic Diabetes Course Cover?

A selected list of questions covered in the Diabetes Course:

  1. What is insulin?
  2. What is insulin toxicity?
  3. How do weight gain and obesity develop?
  4. What is Diabetes?
  5. Can insulin toxicity be reversed?
  6. Can diabetes be reversed?
  7. Is a biology degree necessary for taking Dr. Ali’s Insulin Course and Dr. Ali’s Diabetes Course? The answer: No.

 


Question: Who Should Consider Basic Insulin and Diabetes Courses?

  1. People interested in health and healing.
  2. Parents interested in the health of their children, especially obesity, diabetes, and healthy living.
  3. Teachers teaching school and college classes.
  4. Healthy study groups in communities, associations, at

 Answer: Teachers teaching school health classes.

 


 

Question: Who Should Consider Intermediate Insulin and Diabetes Courses?

Answer: Teachers who teach college-level nutrition and health classes

Anyone whose natural curiosity and interest about the subjects of health, healing, insulin toxicity, weight gain, obesity, and reversal of diabetes has been sharpened by the basic couse.


 Question: Who Should Consider Advanced Insulin and Diabetes Courses?

Doctors and professors who teach advanced health, nutrition, and diabetes classes.

Those and who are iinsulin toxic or has diabetes and who wishes to clear insulin toxicity or has Type 2 diabetes and wishes to reverse the disease.


 Learning and Teaching Materials

  1. Video Seminars: Dr. Ali’s Insulin Toxicity and Diabetes Courses 7 Video seminars (This is Seminar One).
  2. Books: Dr. Ali’s Diabetes Reversal Plan
  3. Courses Taught by Dr. Ali Himself (call 212-873-2444 for course info.

 

Oxygen Model of Diabetes

 

My Oxygen Model of Diabetes is an extension of my Oxygen Model of Health and Disease. It is a unifying model that explains all aspects of Type 2 diabetes ( the type that affects more than 95% of individuals afflicted by diabetest—causes, clinical course, consequences, and control—on the basis of disturbed oxygen function. The most important among these compromised and/or blocked functions are: (1) oxygen signaling; (2) oxygen’s ATP energy generation; (3) oxygen’s detergent functions; (4) oxygen’s cellular detox functions; (5) oxygen-regulated cell membrane and matrix functions; (6) oxygen’s cellular repair roles.

The Oxygen Model of Diabetes provides a simple model that allows physicians to reduce complexities of diverse clinical syndromes into a workable simplicity.

 

This model predicts that ongoing research will reveal that components of acidosis (excess acidity), oxidosis (increased oxidative stress), and CUD (clotting-unclotting dysequilibrium) will be found to play important roles in the pathology and clinical features of Type 2 diabetes.


The crucial importance of  the Unifying Oxygen Model of Type diabetes is that it:

☞ Explains the scientific basis of Type 2 diabetes and its complications;

☞ Sheds light how Type 2 diabetes can be prevented and reversed by addressing all oxygen-related issues;

☞ Elucidates how toxicities of foods, environments, and thoughts cause tissue injury and lead to Type 2 diabetes;

☞ Reveals the mechanisms by which various detox therapies work (Oxygen is the primal detergent which removes cellular grease and allows cells to breathe freely); and

☞ Allows the formulation of rational and effective designs for reversing Type 2 diabetes; and

☞ Provides explanations of mechanisms by which  time-honored natural remedies work to control and prevent Type 2 diabetes.

☞ Provides explanations of mechanisms by which  time-honored natural

 

WHY IS DIAGNOSIS OF DIABETES DELAYED IN MOST PEOPLE?

 

Majid Ali, M.D.

Because insulin tests are delayed, often for years. 


In general, fasting Blood sugar test is relied upon for screening for diabetes.  I have seen many cases in which diabetes remained undiagnosed because the fasting blood sugar was below 100 mg/dL. Had a complete three-hour insulin test been done sooner, the diagnosis of diabetes would have been readily made.

 

Similarly, A1c test is not a reliable test for diagnosis. I have seen this test to also miss diabetes diagnosis. Again,  if three-hour insulin test had been done sooner, the diagnosis of diabetes would have been readily made.


 

For more info, consider my following free courses on this web site:

  1. Dr. Ali’s Diabetes Course
  2. Dr. Ali’s Insulin Course

 

DIABETES IS A SUGAR PROBLEM, NOT AN INSULIN PROBLEM

Majid Ali, M.D.

Some readers have been dismayed by my statement that diabetes is not a sugar problem.


Below are some questions they have raised. My answers follow the questions.

CHALLENGES

  1. Ali’s is wrong. Everyone in the world knows that diabetes is a sugar problem,” one dissenter complained.
  2. How can Dr. Ali be wrong and all other doctors in the world be wrong on the sugar-diabetes question?” another asked.
  3. Doesn’t he know that blood sugar is high in diabetes? Why would doctors do blood testing if diabetes was not a sugar problem?
  4. Doctors do blood A1c test. Isn’t it a sugar test,” comes another challenge.

RESPONSES

  1. Everyone in the world knows that diabetes is a sugar problem. This not true. Scientists and well-informed doctors know that excess insulin (insulin toxicity) predates diabetes by five, ten, or more years.
  2. All doctors in the world would be wrong on the sugar-diabetes question. No, all doctors would not be wrong. I have never met any doctor who denies the scientific facts that insulin toxicity (hyperinsulinism) predate Type 2 diabetes (the type of diabetes that affects more than 90% of diabetics in the world).
  3. Does Dr. Ali know that blood sugar is high in diabetes? Yes, I know that. But blood sugar begins to rise years after blood insulin levels rise and begin to injure various body organs. I explain my simple point below with a kitchen gas rang” analogy.
  4. Isn’t A1c test a test for blood sugar? Yes, it is, but this test is not a reliable test for screening for diagnosing diabetes. I have seen patients in which A1c is in the normal range and the patients has diabetes, and other patients whose A1c value is higher but a three-hour test for glucose does not sjows evidence of diabetes.

KITCHEN GAS STOVE ANALOGY

Let us suppose that the gas line in the basement of a house leaks for weeks and then one day there is a fire in the kitchen stove and the house burns down. What would we blame for the house fire, the leaking gas line in the basement or the kitchen stove?

INSULIN TOXICITY BY MANY NAMES


Majid Ali, M.D.

Simple Insulin Truths

Keep insulin low without drugs and live longer,
or keep blood sugar low with drugs and die young.


 

 

The global tide of diabetes cannot be understood and stemmed without knowing the fundamentals of the molecular biology of insulin, a subject that is sadly and regularly neglected in the prevailing model of medicine. I wrote Insulin Toxicity Series to shed light on the various faces of this pandemic.


Seven Faces of insulin toxicity:

☞ The first stage of insulin toxicity is without apparent negative health effects recognized by the person.
☞ The second stage of insulin toxicity is with negative health effects recognized by the person but unknown to the doctor.
☞ The third stage of insulin toxicity is tissue injury (in the liver, kidneys, skin, and other organs) unrecognized by a doctor who is clueless about molecular biology of insulin.
☞ The fourth stage of insulin toxicity is prediabetes without tests to detect insulin waste and damages.
☞ The fifth stage of insulin toxicity is Type 2 diabetes with the use of diabetes drugs that add to the insulin activity, hence its toxicity, with the blessings of the American Diabetic Association).
☞ The sixth stage of insulin toxicity is toxicity created by peaks of insulin caused by insulin injections (the end-stage of insulin depletion which is called insulin-dependent diabetes).
☞ The seventh stage of insulin toxicity is loss of vision and blindness (diabetic retinopathy), dialysis (diabetic nephropathy), and increased risk of heart attacks, strokes, autoimmune diseases, inflammatory disorders, and all degenerative states.

I present evidence for all of the above in other articles in my Insulin Toxicity Series:

☞ The Crank and Crank-shaft Model of Insulin Toxicity
☞ The Evidence for the Grease and Detergent Model of Insulin Toxicity
☞ Seven Stages of Insulin Toxicity
☞ Insulin and Metabolic Frugality
☞ Saving the Unborn from Maternal Insulin Toxicity
☞ Insulin-Aging Connections
☞ The Insulin-Sugar Connections
☞ The Insulin-Lipid Connections
☞ The Insulin-Fat Cell Connections (The Intelligent Fat Cells)
☞ The Insulin-Protein Connections
☞ The Insulin-Gut Connections
☞ The Insulin-Liver Connections
☞ The Insulin-Heart Connections
☞ The Insulin-Obesity Connections
☞ The Insulin-Kidney Connections
☞ The Insulin-Brain Connections
☞ The Insulin-Ovary Connections
☞ The Insulin-Eye Connections
☞ Insulin Toxicity and Metformin Mindlessness

The take-home message of this article is: the use of diabetes drugs to lower blood sugar levels without non-drug plans to lower blood insulin levels is feeding the pandemic of insulin toxicity and diabetes. The American Diabetic Association and The New England Journal of Medicine teach doctors to use only drugs to lower blood sugar when what the people really need are programs to de-grease the cell membranes, free up insulin receptor proteins embedded in the membranes, and lower blood insulin levels.

The last article entitled “Insulin Toxicity and Metformin Mindlessness” reveals how many holistic doctors are succumbing to the lure of easy answers with drugs that do not address the real issues.

 


 

Dr. Ali’s Insulin Video Library


Google Search for Dr. Ali’s Insulin Library

bout 47,900 results

 

 

Why Is the pancreas gland that produces insulin so low in the order of body organs?

I invite readers to keep this question in the mind as they consider Dr. Ali’s Course on Diabetes Reversal.


 

 

My Free Diabetes Course Has Two Parts: (1) Part One – Dr. Ali’s Insulin Toxicity Course; and (2) Part Two: Dr. Ali’s Diabetes Reversal Course. The first part of the course concerns the problem and the second part the solution. 


Scientific Basis of Insulin-Based Diabetes Reversal

Scientific Basis of Dr. Ali’s Diabetes Insulin Toxicity and Diabetes Reversal Courses

  1. Molecular Biology of Oxygen
  2. Insulin homeostasis

Five Threats to Humankind:

  1. Developmental Challenges of the Unborn
  2. Diabetes
  3. Dialysis
  4. Dementia
  5. Disability

All  five are rooted in insulin toxicity. I anticipate that some readers will roll their eyes on the first item listed above. That only means they are not aware of the frequency with which hyperinsulinism is encountered in children with autism, dysautonomia, OCD, POTS, and related neurodevelopmental challenges faced by children in prenatal and postnatal lives.


 

Oxygen Models of Insulin Toxicity and Diabetes Reversal Five Threats to Humankind:

Dr. Ali’s Insulin Toxicity Course and Dr. Ali’s Diabetes Reversal Course are based on Oxygen Models of Diabetes and Insulin Toxicity. Simply stated, these models explains all aspects of Type 2 diabetes—causes, clinical course, consequences, and control—on the basis of disturbed oxygen function. A full description of these models is included at the end of this article.


Body Organs of Special Interest in Oxygen Models of Diabetes and Insulin Toxicity

  1. Gut
  2. Liver
  3. Thalamus in the Brain
  4. Muscles
  5. Pancreas

Why Is the pancreas gland that produces insulin so low in the order of body organs?

I invite readers to keep this question in the mind as they consider my Course on Diabetes?


 

The Gut-Diabetes Connection

 The Gut-Diabetes Connections

  • Throat
  • Esophagus
  • Stomach
  • Small intestine
  • Large intestine

Digestion starts within the mouth by the action of the enzymes in saliva. It then takes full effect within the stomach and some nutrients are also absorbed into the bloodstream here. Partially digested food known as chyme then undergoes further digestion mainly in the first part of the small intestine known as the duodenum. The small intestine, or small bowel, is the longest part of the gut and gradually the food is completely digested and almost all the nutrients are absorbed into the bloodstream.


The Thalamus-Feeding-Weight-Diabetes Connections

Picture

The Quick Facts

Location: Part of the forebrain, below the corpus callosum
Function: Responsible for relaying information from the sensory receptors to proper areas of the brain where it can be processed
The thalamus in the brain has special centers for glucose and regulates some aspects of  sensory information that is being transmitted to the brain.
 

 

What Is More Important in Diabetes?

In Beta Cells of the pancreas where insulin is produced?

Or in cell membranes where it moves receptor proteins?

muscleWhere Insulin Is Produced Or Where It Is Used?


What Is Insulin? Where Does It Come From?

The pancreas is a long, slender organ, most of which is located posterior to the bottom half of the stomach.  Although it is primarily an exocrine gland, secreting a variety of digestive enzymes, the pancreas has an endocrine function. Its pancreatic islets—clusters of cells formerly known as the islets of Langerhans—secrete the hormones glucagon, insulin, somatostatin, and pancreatic polypeptide (PP).

Pancreas

This diagram shows the anatomy of the pancreas. The left, larger side of the pancreas is seated within the curve of the duodenum of the small intestine. The smaller, rightmost tip of the pancreas is located near the spleen. The splenic artery is seen travelling to the spleen, however, it has several branches connecting to the pancreas. An interior view of the pancreas shows that the pancreatic duct is a large tube running through the center of the pancreas. It branches throughout its length in to several horseshoe- shaped pockets of acinar cells. These cells secrete digestive enzymes, which travel down the bile duct and into the small intestine. There are also small pancreatic islets scattered throughout the pancreas. The pancreatic islets secrete the pancreatic hormones insulin and glucagon into the splenic artery. An inset micrograph shows that the pancreatic islets are small discs of tissue consisting of a thin, outer ring called the exocrine acinus, a thicker, inner ring of beta cells and a central circle of alpha cells.

The pancreatic exocrine function involves the acinar cells secreting digestive enzymes that are transported into the small intestine by the pancreatic duct. Its endocrine function involves the secretion of insulin (produced by beta cells) and glucagon 

Two Dimensions of Dr. Ali’s Diabetes Course for Reversing Type 2 Diabetes

          ☞ Insulin Toxicity Course (to Know the Problem Well

         ☞ Diabetes Reversal Course to Know then Solution Well for Reversing Diabetes?

 

I offer my course in two parts: (1) Dr. Ali’s insulin Toxicity Course; and (2) Dr. Ali’s Diabetes Reversal Course. I attribute the two parts of this course to myself for the simple reason that it makes it easier for people to find it on the internet.

Dr. Ali’s Diabetes Course and Insulin Toxicity Courses are free for everyone, and are posted athttp://www.alidiabetes.org. For my free recipes, please go to http://www.alidiabetes.org. 

Should anyone or any institution wish to teach this course, please send me a note and I will send you written permission to do so without any cost.


Dt. Ali’s Basic, Intermediate, and Advanced Diabetes Courses

 My both Diabetes Course and Insulin Toxicity Course are subdivided into three levels as follows:

  1. Ali’s Basic Diabetes Course
  2. Ali’s Intermediate Diabetes Course
  3. Ali’s Advanced Diabetes Course
  4. Ali’s Basic Insulin Toxicity Course
  5. Ali’s Intermediate Insulin Toxicity Course
  6. Ali’s Advanced Insulin Toxicity Course

What Does the Basic Diabetes Course Cover?

A selected list of questions covered in the Diabetes Course:

  1. What is insulin?
  2. What is insulin toxicity?
  3. How do weight gain and obesity develop?
  4. What is Diabetes?
  5. Can insulin toxicity be reversed?
  6. Can diabetes be reversed?
  7. Is a biology degree necessary for taking Dr. Ali’s Insulin Course and Dr. Ali’s Diabetes Course? The answer: No.

 


Question: Who Should Consider Basic Insulin and Diabetes Courses?

  1. People interested in health and healing.
  2. Parents interested in the health of their children, especially obesity, diabetes, and healthy living.
  3. Teachers teaching school and college classes.
  4. Healthy study groups in communities, associations, at

 Answer: Teachers teaching school health classes.

 


 

Question: Who Should Consider Intermediate Insulin and Diabetes Courses?

Answer: Teachers who teach college-level nutrition and health classes

Anyone whose natural curiosity and interest about the subjects of health, healing, insulin toxicity, weight gain, obesity, and reversal of diabetes has been sharpened by the basic couse.


 Question: Who Should Consider Advanced Insulin and Diabetes Courses?

Doctors and professors who teach advanced health, nutrition, and diabetes classes.

Those and who are iinsulin toxic or has diabetes and who wishes to clear insulin toxicity or has Type 2 diabetes and wishes to reverse the disease.


 Learning and Teaching Materials

  1. Video Seminars: Dr. Ali’s Insulin Toxicity and Diabetes Courses 7 Video seminars (This is Seminar One).
  2. Books: Dr. Ali’s Diabetes Reversal Plan
  3. Courses Taught by Dr. Ali Himself (call 212-873-2444 for course info.

 

Oxygen Model of Diabetes

 

My Oxygen Model of Diabetes is an extension of my Oxygen Model of Health and Disease. It is a unifying model that explains all aspects of Type 2 diabetes ( the type that affects more than 95% of individuals afflicted by diabetest—causes, clinical course, consequences, and control—on the basis of disturbed oxygen function. The most important among these compromised and/or blocked functions are: (1) oxygen signaling; (2) oxygen’s ATP energy generation; (3) oxygen’s detergent functions; (4) oxygen’s cellular detox functions; (5) oxygen-regulated cell membrane and matrix functions; (6) oxygen’s cellular repair roles.

The Oxygen Model of Diabetes provides a simple model that allows physicians to reduce complexities of diverse clinical syndromes into a workable simplicity.

 

This model predicts that ongoing research will reveal that components of acidosis (excess acidity), oxidosis (increased oxidative stress), and CUD (clotting-unclotting dysequilibrium) will be found to play important roles in the pathology and clinical features of Type 2 diabetes.


The crucial importance of  the Unifying Oxygen Model of Type diabetes is that it:

☞ Explains the scientific basis of Type 2 diabetes and its complications;

☞ Sheds light how Type 2 diabetes can be prevented and reversed by addressing all oxygen-related issues;

☞ Elucidates how toxicities of foods, environments, and thoughts cause tissue injury and lead to Type 2 diabetes;

☞ Reveals the mechanisms by which various detox therapies work (Oxygen is the primal detergent which removes cellular grease and allows cells to breathe freely); and

☞ Allows the formulation of rational and effective designs for reversing Type 2 diabetes; and

☞ Provides explanations of mechanisms by which  time-honored natural remedies work to control and prevent Type 2 diabetes.

☞ Provides explanations of mechanisms by which  time-honored natural

 

WHY IS DIAGNOSIS OF DIABETES DELAYED IN MOST PEOPLE?

 

Majid Ali, M.D.

Because insulin tests are delayed, often for years. 


In general, fasting Blood sugar test is relied upon for screening for diabetes.  I have seen many cases in which diabetes remained undiagnosed because the fasting blood sugar was below 100 mg/dL. Had a complete three-hour insulin test been done sooner, the diagnosis of diabetes would have been readily made.

 

Similarly, A1c test is not a reliable test for diagnosis. I have seen this test to also miss diabetes diagnosis. Again,  if three-hour insulin test had been done sooner, the diagnosis of diabetes would have been readily made.


 

For more info, consider my following free courses on this web site:

  1. Dr. Ali’s Diabetes Course
  2. Dr. Ali’s Insulin Course

 

DIABETES IS A SUGAR PROBLEM, NOT AN INSULIN PROBLEM

Majid Ali, M.D.

Some readers have been dismayed by my statement that diabetes is not a sugar problem.


Below are some questions they have raised. My answers follow the questions.

CHALLENGES

  1. Ali’s is wrong. Everyone in the world knows that diabetes is a sugar problem,” one dissenter complained.
  2. How can Dr. Ali be wrong and all other doctors in the world be wrong on the sugar-diabetes question?” another asked.
  3. Doesn’t he know that blood sugar is high in diabetes? Why would doctors do blood testing if diabetes was not a sugar problem?
  4. Doctors do blood A1c test. Isn’t it a sugar test,” comes another challenge.

RESPONSES

  1. Everyone in the world knows that diabetes is a sugar problem. This not true. Scientists and well-informed doctors know that excess insulin (insulin toxicity) predates diabetes by five, ten, or more years.
  2. All doctors in the world would be wrong on the sugar-diabetes question. No, all doctors would not be wrong. I have never met any doctor who denies the scientific facts that insulin toxicity (hyperinsulinism) predate Type 2 diabetes (the type of diabetes that affects more than 90% of diabetics in the world).
  3. Does Dr. Ali know that blood sugar is high in diabetes? Yes, I know that. But blood sugar begins to rise years after blood insulin levels rise and begin to injure various body organs. I explain my simple point below with a kitchen gas rang” analogy.
  4. Isn’t A1c test a test for blood sugar? Yes, it is, but this test is not a reliable test for screening for diagnosing diabetes. I have seen patients in which A1c is in the normal range and the patients has diabetes, and other patients whose A1c value is higher but a three-hour test for glucose does not sjows evidence of diabetes.

KITCHEN GAS STOVE ANALOGY

Let us suppose that the gas line in the basement of a house leaks for weeks and then one day there is a fire in the kitchen stove and the house burns down. What would we blame for the house fire, the leaking gas line in the basement or the kitchen stove?

INSULIN TOXICITY BY MANY NAMES


Majid Ali, M.D.

Simple Insulin Truths

Keep insulin low without drugs and live longer,
or keep blood sugar low with drugs and die young.


 

 

The global tide of diabetes cannot be understood and stemmed without knowing the fundamentals of the molecular biology of insulin, a subject that is sadly and regularly neglected in the prevailing model of medicine. I wrote Insulin Toxicity Series to shed light on the various faces of this pandemic.


Seven Faces of insulin toxicity:

☞ The first stage of insulin toxicity is without apparent negative health effects recognized by the person.
☞ The second stage of insulin toxicity is with negative health effects recognized by the person but unknown to the doctor.
☞ The third stage of insulin toxicity is tissue injury (in the liver, kidneys, skin, and other organs) unrecognized by a doctor who is clueless about molecular biology of insulin.
☞ The fourth stage of insulin toxicity is prediabetes without tests to detect insulin waste and damages.
☞ The fifth stage of insulin toxicity is Type 2 diabetes with the use of diabetes drugs that add to the insulin activity, hence its toxicity, with the blessings of the American Diabetic Association).
☞ The sixth stage of insulin toxicity is toxicity created by peaks of insulin caused by insulin injections (the end-stage of insulin depletion which is called insulin-dependent diabetes).
☞ The seventh stage of insulin toxicity is loss of vision and blindness (diabetic retinopathy), dialysis (diabetic nephropathy), and increased risk of heart attacks, strokes, autoimmune diseases, inflammatory disorders, and all degenerative states.

I present evidence for all of the above in other articles in my Insulin Toxicity Series:

☞ The Crank and Crank-shaft Model of Insulin Toxicity
☞ The Evidence for the Grease and Detergent Model of Insulin Toxicity
☞ Seven Stages of Insulin Toxicity
☞ Insulin and Metabolic Frugality
☞ Saving the Unborn from Maternal Insulin Toxicity
☞ Insulin-Aging Connections
☞ The Insulin-Sugar Connections
☞ The Insulin-Lipid Connections
☞ The Insulin-Fat Cell Connections (The Intelligent Fat Cells)
☞ The Insulin-Protein Connections
☞ The Insulin-Gut Connections
☞ The Insulin-Liver Connections
☞ The Insulin-Heart Connections
☞ The Insulin-Obesity Connections
☞ The Insulin-Kidney Connections
☞ The Insulin-Brain Connections
☞ The Insulin-Ovary Connections
☞ The Insulin-Eye Connections
☞ Insulin Toxicity and Metformin Mindlessness

The take-home message of this article is: the use of diabetes drugs to lower blood sugar levels without non-drug plans to lower blood insulin levels is feeding the pandemic of insulin toxicity and diabetes. The American Diabetic Association and The New England Journal of Medicine teach doctors to use only drugs to lower blood sugar when what the people really need are programs to de-grease the cell membranes, free up insulin receptor proteins embedded in the membranes, and lower blood insulin levels.

The last article entitled “Insulin Toxicity and Metformin Mindlessness” reveals how many holistic doctors are succumbing to the lure of easy answers with drugs that do not address the real issues.

 


 

Dr. Ali’s Insulin Video Library


Google Search for Dr. Ali’s Insulin Library

bout 47,900 results

 

FOR DIABETES, IS PANCREAS MORE IMPORTANT OR THE GUT?

Majid Ali, M.D.

Some doctors might find the question annoying. And that is so because they do not do insulin testing for their patients.


Body Organs of Special Interest in Oxygen Models of Diabetes and Insulin Toxicity

  1. Gut
  2. Liver
  3. Thalamus in the Brain
  4. Muscles
  5. Pancreas

Why Is the pancreas gland that produces insulin so low in the order of body organs?

I invite readers to keep this question in the mind as they consider Dr. Ali’s Course on Diabetes Reversal.


 

For full explanation of my answer, please read more at Dr. Ali’s Diabetes Course by clicking at the link below.

.

 

DR. ALI’S DIABETES COURSE

 

Majid Ali, M.D.

My Free Diabetes Course Has Two Parts: (1) Part One – Dr. Ali’s Insulin Toxicity Course; and (2) Part Two: Dr. Ali’s Diabetes Reversal Course. The first part of the course concerns the problem and the second part the solution. 


Scientific Basis of Insulin-Based Diabetes Reversal

Scientific Basis of Dr. Ali’s Diabetes Insulin Toxicity and Diabetes Reversal Courses

  1. Molecular Biology of Oxygen
  2. Insulin homeostasis

Five Threats to Humankind:

  1. Developmental Challenges of the Unborn
  2. Diabetes
  3. Dialysis
  4. Dementia
  5. Disability

All  five are rooted in insulin toxicity. I anticipate that some readers will roll their eyes on the first item listed above. That only means they are not aware of the frequency with which hyperinsulinism is encountered in children with autism, dysautonomia, OCD, POTS, and related neurodevelopmental challenges faced by children in prenatal and postnatal lives.


 

Oxygen Models of Insulin Toxicity and Diabetes Reversal Five Threats to Humankind:

Dr. Ali’s Insulin Toxicity Course and Dr. Ali’s Diabetes Reversal Course are based on Oxygen Models of Diabetes and Insulin Toxicity. Simply stated, these models explains all aspects of Type 2 diabetes—causes, clinical course, consequences, and control—on the basis of disturbed oxygen function. A full description of these models is included at the end of this article.


Body Organs of Special Interest in Oxygen Models of Diabetes and Insulin Toxicity

  1. Gut
  2. Liver
  3. Thalamus in the Brain
  4. Muscles
  5. Pancreas

Why Is the pancreas gland that produces insulin so low in the order of body organs?

I invite readers to keep this question in the mind as they consider my Course on Diabetes?


 

The Gut-Diabetes Connection

 The Gut-Diabetes Connections

  • Throat
  • Esophagus
  • Stomach
  • Small intestine
  • Large intestine

Digestion starts within the mouth by the action of the enzymes in saliva. It then takes full effect within the stomach and some nutrients are also absorbed into the bloodstream here. Partially digested food known as chyme then undergoes further digestion mainly in the first part of the small intestine known as the duodenum. The small intestine, or small bowel, is the longest part of the gut and gradually the food is completely digested and almost all the nutrients are absorbed into the bloodstream.


The Thalamus-Feeding-Weight-Diabetes Connections

Picture

The Quick Facts

Location: Part of the forebrain, below the corpus callosum
Function: Responsible for relaying information from the sensory receptors to proper areas of the brain where it can be processed
The thalamus in the brain has special centers for glucose and regulates some aspects of  sensory information that is being transmitted to the brain.
 

 

What Is More Important in Diabetes?

In Beta Cells of the pancreas where insulin is produced?

Or in cell membranes where it moves receptor proteins?

muscleWhere Insulin Is Produced Or Where It Is Used?


What Is Insulin? Where Does It Come From?

The pancreas is a long, slender organ, most of which is located posterior to the bottom half of the stomach.  Although it is primarily an exocrine gland, secreting a variety of digestive enzymes, the pancreas has an endocrine function. Its pancreatic islets—clusters of cells formerly known as the islets of Langerhans—secrete the hormones glucagon, insulin, somatostatin, and pancreatic polypeptide (PP).

Pancreas

This diagram shows the anatomy of the pancreas. The left, larger side of the pancreas is seated within the curve of the duodenum of the small intestine. The smaller, rightmost tip of the pancreas is located near the spleen. The splenic artery is seen travelling to the spleen, however, it has several branches connecting to the pancreas. An interior view of the pancreas shows that the pancreatic duct is a large tube running through the center of the pancreas. It branches throughout its length in to several horseshoe- shaped pockets of acinar cells. These cells secrete digestive enzymes, which travel down the bile duct and into the small intestine. There are also small pancreatic islets scattered throughout the pancreas. The pancreatic islets secrete the pancreatic hormones insulin and glucagon into the splenic artery. An inset micrograph shows that the pancreatic islets are small discs of tissue consisting of a thin, outer ring called the exocrine acinus, a thicker, inner ring of beta cells and a central circle of alpha cells.

The pancreatic exocrine function involves the acinar cells secreting digestive enzymes that are transported into the small intestine by the pancreatic duct. Its endocrine function involves the secretion of insulin (produced by beta cells) and glucagon 

Two Dimensions of Dr. Ali’s Diabetes Course for Reversing Type 2 Diabetes

          ☞ Insulin Toxicity Course (to Know the Problem Well

         ☞ Diabetes Reversal Course to Know then Solution Well for Reversing Diabetes?

 

I offer my course in two parts: (1) Dr. Ali’s insulin Toxicity Course; and (2) Dr. Ali’s Diabetes Reversal Course. I attribute the two parts of this course to myself for the simple reason that it makes it easier for people to find it on the internet.

Dr. Ali’s Diabetes Course and Insulin Toxicity Courses are free for everyone, and are posted athttp://www.alidiabetes.org. For my free recipes, please go to http://www.alidiabetes.org. 

Should anyone or any institution wish to teach this course, please send me a note and I will send you written permission to do so without any cost.


Dt. Ali’s Basic, Intermediate, and Advanced Diabetes Courses

 My both Diabetes Course and Insulin Toxicity Course are subdivided into three levels as follows:

  1. Ali’s Basic Diabetes Course
  2. Ali’s Intermediate Diabetes Course
  3. Ali’s Advanced Diabetes Course
  4. Ali’s Basic Insulin Toxicity Course
  5. Ali’s Intermediate Insulin Toxicity Course
  6. Ali’s Advanced Insulin Toxicity Course

What Does the Basic Diabetes Course Cover?

A selected list of questions covered in the Diabetes Course:

  1. What is insulin?
  2. What is insulin toxicity?
  3. How do weight gain and obesity develop?
  4. What is Diabetes?
  5. Can insulin toxicity be reversed?
  6. Can diabetes be reversed?
  7. Is a biology degree necessary for taking Dr. Ali’s Insulin Course and Dr. Ali’s Diabetes Course? The answer: No.

 


Question: Who Should Consider Basic Insulin and Diabetes Courses?

  1. People interested in health and healing.
  2. Parents interested in the health of their children, especially obesity, diabetes, and healthy living.
  3. Teachers teaching school and college classes.
  4. Healthy study groups in communities, associations, at

 Answer: Teachers teaching school health classes.

 


 

Question: Who Should Consider Intermediate Insulin and Diabetes Courses?

Answer: Teachers who teach college-level nutrition and health classes

Anyone whose natural curiosity and interest about the subjects of health, healing, insulin toxicity, weight gain, obesity, and reversal of diabetes has been sharpened by the basic couse.


 Question: Who Should Consider Advanced Insulin and Diabetes Courses?

Doctors and professors who teach advanced health, nutrition, and diabetes classes.

Those and who are iinsulin toxic or has diabetes and who wishes to clear insulin toxicity or has Type 2 diabetes and wishes to reverse the disease.


 Learning and Teaching Materials

  1. Video Seminars: Dr. Ali’s Insulin Toxicity and Diabetes Courses 7 Video seminars (This is Seminar One).
  2. Books: Dr. Ali’s Diabetes Reversal Plan
  3. Courses Taught by Dr. Ali Himself (call 212-873-2444 for course info.

 

Oxygen Model of Diabetes

 

My Oxygen Model of Diabetes is an extension of my Oxygen Model of Health and Disease. It is a unifying model that explains all aspects of Type 2 diabetes ( the type that affects more than 95% of individuals afflicted by diabetest—causes, clinical course, consequences, and control—on the basis of disturbed oxygen function. The most important among these compromised and/or blocked functions are: (1) oxygen signaling; (2) oxygen’s ATP energy generation; (3) oxygen’s detergent functions; (4) oxygen’s cellular detox functions; (5) oxygen-regulated cell membrane and matrix functions; (6) oxygen’s cellular repair roles.

The Oxygen Model of Diabetes provides a simple model that allows physicians to reduce complexities of diverse clinical syndromes into a workable simplicity.

 

This model predicts that ongoing research will reveal that components of acidosis (excess acidity), oxidosis (increased oxidative stress), and CUD (clotting-unclotting dysequilibrium) will be found to play important roles in the pathology and clinical features of Type 2 diabetes.


The crucial importance of  the Unifying Oxygen Model of Type diabetes is that it:

☞ Explains the scientific basis of Type 2 diabetes and its complications;

☞ Sheds light how Type 2 diabetes can be prevented and reversed by addressing all oxygen-related issues;

☞ Elucidates how toxicities of foods, environments, and thoughts cause tissue injury and lead to Type 2 diabetes;

☞ Reveals the mechanisms by which various detox therapies work (Oxygen is the primal detergent which removes cellular grease and allows cells to breathe freely); and

☞ Allows the formulation of rational and effective designs for reversing Type 2 diabetes; and

☞ Provides explanations of mechanisms by which  time-honored natural remedies work to control and prevent Type 2 diabetes.

☞ Provides explanations of mechanisms by which  time-honored natural

 

WHY IS DIAGNOSIS OF DIABETES DELAYED IN MOST PEOPLE?

 

Majid Ali, M.D.

Because insulin tests are delayed, often for years. 


In general, fasting Blood sugar test is relied upon for screening for diabetes.  I have seen many cases in which diabetes remained undiagnosed because the fasting blood sugar was below 100 mg/dL. Had a complete three-hour insulin test been done sooner, the diagnosis of diabetes would have been readily made.

 

Similarly, A1c test is not a reliable test for diagnosis. I have seen this test to also miss diabetes diagnosis. Again,  if three-hour insulin test had been done sooner, the diagnosis of diabetes would have been readily made.


 

For more info, consider my following free courses on this web site:

  1. Dr. Ali’s Diabetes Course
  2. Dr. Ali’s Insulin Course

 

DIABETES IS A SUGAR PROBLEM, NOT AN INSULIN PROBLEM

Majid Ali, M.D.

Some readers have been dismayed by my statement that diabetes is not a sugar problem.


Below are some questions they have raised. My answers follow the questions.

CHALLENGES

  1. Ali’s is wrong. Everyone in the world knows that diabetes is a sugar problem,” one dissenter complained.
  2. How can Dr. Ali be wrong and all other doctors in the world be wrong on the sugar-diabetes question?” another asked.
  3. Doesn’t he know that blood sugar is high in diabetes? Why would doctors do blood testing if diabetes was not a sugar problem?
  4. Doctors do blood A1c test. Isn’t it a sugar test,” comes another challenge.

RESPONSES

  1. Everyone in the world knows that diabetes is a sugar problem. This not true. Scientists and well-informed doctors know that excess insulin (insulin toxicity) predates diabetes by five, ten, or more years.
  2. All doctors in the world would be wrong on the sugar-diabetes question. No, all doctors would not be wrong. I have never met any doctor who denies the scientific facts that insulin toxicity (hyperinsulinism) predate Type 2 diabetes (the type of diabetes that affects more than 90% of diabetics in the world).
  3. Does Dr. Ali know that blood sugar is high in diabetes? Yes, I know that. But blood sugar begins to rise years after blood insulin levels rise and begin to injure various body organs. I explain my simple point below with a kitchen gas rang” analogy.
  4. Isn’t A1c test a test for blood sugar? Yes, it is, but this test is not a reliable test for screening for diagnosing diabetes. I have seen patients in which A1c is in the normal range and the patients has diabetes, and other patients whose A1c value is higher but a three-hour test for glucose does not sjows evidence of diabetes.

KITCHEN GAS STOVE ANALOGY

Let us suppose that the gas line in the basement of a house leaks for weeks and then one day there is a fire in the kitchen stove and the house burns down. What would we blame for the house fire, the leaking gas line in the basement or the kitchen stove?

INSULIN TOXICITY BY MANY NAMES


Majid Ali, M.D.

Simple Insulin Truths

Keep insulin low without drugs and live longer,
or keep blood sugar low with drugs and die young.


 

 

The global tide of diabetes cannot be understood and stemmed without knowing the fundamentals of the molecular biology of insulin, a subject that is sadly and regularly neglected in the prevailing model of medicine. I wrote Insulin Toxicity Series to shed light on the various faces of this pandemic.


Seven Faces of insulin toxicity:

☞ The first stage of insulin toxicity is without apparent negative health effects recognized by the person.
☞ The second stage of insulin toxicity is with negative health effects recognized by the person but unknown to the doctor.
☞ The third stage of insulin toxicity is tissue injury (in the liver, kidneys, skin, and other organs) unrecognized by a doctor who is clueless about molecular biology of insulin.
☞ The fourth stage of insulin toxicity is prediabetes without tests to detect insulin waste and damages.
☞ The fifth stage of insulin toxicity is Type 2 diabetes with the use of diabetes drugs that add to the insulin activity, hence its toxicity, with the blessings of the American Diabetic Association).
☞ The sixth stage of insulin toxicity is toxicity created by peaks of insulin caused by insulin injections (the end-stage of insulin depletion which is called insulin-dependent diabetes).
☞ The seventh stage of insulin toxicity is loss of vision and blindness (diabetic retinopathy), dialysis (diabetic nephropathy), and increased risk of heart attacks, strokes, autoimmune diseases, inflammatory disorders, and all degenerative states.

I present evidence for all of the above in other articles in my Insulin Toxicity Series:

☞ The Crank and Crank-shaft Model of Insulin Toxicity
☞ The Evidence for the Grease and Detergent Model of Insulin Toxicity
☞ Seven Stages of Insulin Toxicity
☞ Insulin and Metabolic Frugality
☞ Saving the Unborn from Maternal Insulin Toxicity
☞ Insulin-Aging Connections
☞ The Insulin-Sugar Connections
☞ The Insulin-Lipid Connections
☞ The Insulin-Fat Cell Connections (The Intelligent Fat Cells)
☞ The Insulin-Protein Connections
☞ The Insulin-Gut Connections
☞ The Insulin-Liver Connections
☞ The Insulin-Heart Connections
☞ The Insulin-Obesity Connections
☞ The Insulin-Kidney Connections
☞ The Insulin-Brain Connections
☞ The Insulin-Ovary Connections
☞ The Insulin-Eye Connections
☞ Insulin Toxicity and Metformin Mindlessness

The take-home message of this article is: the use of diabetes drugs to lower blood sugar levels without non-drug plans to lower blood insulin levels is feeding the pandemic of insulin toxicity and diabetes. The American Diabetic Association and The New England Journal of Medicine teach doctors to use only drugs to lower blood sugar when what the people really need are programs to de-grease the cell membranes, free up insulin receptor proteins embedded in the membranes, and lower blood insulin levels.

The last article entitled “Insulin Toxicity and Metformin Mindlessness” reveals how many holistic doctors are succumbing to the lure of easy answers with drugs that do not address the real issues.

 


 

Dr. Ali’s Insulin Video Library


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