Category Archives: 3D Insulin Protocol

Can You Reverse Diabetes?

Majid Ali, M.D.

Are You Willing and Able to Try? Is So, Continue to Read.


 

Only You Can Answer the Question in the Title.
The information given below can help you. 


Dr. Ali’s Breakfast Shakes

Majid Ali, M.D/ Dr. Ali’s Breakfast Shakes Are Ideas, Not Products   Shakes for Weight Loss and Diabetes Reversal And Related Insulin-Smart Omelettes and Other Insulin-Smart Breakfasts for Insulin-Smart eating There is n…
No Ads with WordPress.com PremiumPrevent ads from showing on your site.

The Insulin Diet

Two Insulin Diet Plans Majid Ali, M.D. Insulin is the hunger hormone. This scientific simplicity forms the core of my Insulin Diet. I  prescribe it for my patients in one of its two forms: 1.             Insulin Diet One…

Insulin-Monitored Diabetes Reversal

Majid Ali, M.D.   Yes, almond butter is an insulin-smart food. Almond butter does not cause a blood sugar spike. So it does not cause blood insulin spike.   Almond butter is a good food also because it is rich in mineral…

The LOPI Way to Reversing Diabetes

Majid Ali, M.D. The Love-Oxygen-Prayer-Insulin (LOPI) Way for Reversing Diabetes Please consider this Path Away From Diabetes for yourself and for those you love. It is not a path of products. I is the path to truth, lov…

Insulin Toxicity of the Unborn

Majid Ali, M.D. The incidence of pregnancy-associated insulin resistance is rising worldwide, I think it is appropriately designated as insulin toxicity of the unborn. The incidence of pregnancy-associated insulin resist…

Optimal and Inappropriate Laboratory Testing For Assessing Insulin Homeostasis

Majid Ali, M.D. Grievous Errors in Insulin Testing   What Is Optimal Laboratory Insulin Testing? What Are Commonly Made Grievous Insulun Testing Errors?  Optimal laboratory testing for assessing insulin homeostasis is to…

Weight Loss – Truths and Mistruths

  Majid Ali, M.D. Another Hormone for Weight Loss and for Not Looking Like a Pear The only honest way of weight loss without losing health is eating less. The scientific truths behind this statement are: Insulin is the f…

Hyperinsulinism Associated With Breast and Prostate Cancer

Majid Ali, M.D. Published in the Journal Townsend Letter (2017;409:66-69 (August 2017)   Hyperinsulinism fans the fire of cancer. In this article, I present case studies to show diet and integrative therapies can restore…

Free Access Diabetes Library

Majid Ali, M.D.


Library of Articles and Videos

Dr. Ali’s Three-Part Diabetes Course
 
Dr. Ali’s Diabetes Course – Part 1: The Basics of Diabetes
https://alidiabetes.org/2016/06/27/dr-alis-diabetes…-part-one-basics/ ‎
 
Dr. Ali’s Diabetes Course – Part 2: Insulin Detox – Beyond Sugar Talk
https://alidiabetes.org/2016/07/11/dr-alis-diabetes-course-part-two-2/ ‎
 
Dr. Ali’s Diabetes Course – Part 3:
https://alidiabetes.org/2016/07/25/dr-alis-3-part-d…ourse-part-three/
 
 
Breakfasts 2016
 
MMM
 
Lab Ref Ranges
 
 
 
Reversing Diabetes Pack
Reversing Diabetes – Lesson One
DR. ALI’S 3-PART DIABETES COURSE PART TWO
DR. ALI’S 3-PART DIABETES COURSE – PART THREE
Reversing Diabetes – Lesson Four
https://alidiabetes.org/2016/08/15/reversing-diabetes-lesson-four/
Reversing Diabetes – Lesson Five
Reversing Diabetes – Lesson Six
Reversing Diabetes – Lesson Seven Spiritual Speak
Reversing Diabetes – Seven Simple Lessons
Diabetes Recipes
DR. ALI’S 3-PART DIABETES COURSE – PART THREE
DR. ALI’S 3-PART DIABETES COURSE PART TWO
alink: https://alidiabetes.org/2016/08/15/reversing-diabetes-lesson-four/ ‎Edit Get

DIABETES VIDEO LIBRARY

Diabetes videos part 1 | The Ali Academy Community

In this 55-minute video seminar, Professor Majid Ali, M.D. discusses the causes, clinical features, and consequences of insulin toxicity, including pre-diabetes …

Diabetes Insulin Videos – Ali Healing Community

Majid Ali, M.D. Links to Videos on Prevent and Reverse Diabetes What is Diabeteshttps://www.youtube.com/watch?v=vTUFY2It-vQ What Is Insulin? What Are Its …

Majid Ali, M.D. * Insulin Toxicity De-mystifies the Metabolic Syndrome …

Jun 28, 2012 – Uploaded by majid ali

The true mature of the metabolic syndrome is insulin toxicity. The term metabolic syndrome creates creates …

Majid Ali, M.D. * Can You Increase Natural Insulin in Diabetes …

Jun 5, 2012 – Uploaded by majid ali

The answer is YES in many cases. I illustrate this with a case study. In advanced stages of diabetes Type 2 …

Majid Ali MD, Castor Oil Rubs for Insulin Detox for Weight Loss and …

https://vimeo.com › Majid Ali › Videos
Jan 24, 2015 – Uploaded by Majid Ali

Type 2 diabetes is an insulin-toxicity state for years before the body reserves ofinsulin are depleted and the …

Majid Ali MD, Dr. Ali’s Book on Reversing Diabetes – Dr. Ali’s Plan for …

https://vimeo.com › Majid Ali › Videos
Nov 6, 2014 – Uploaded by Majid Ali

I outline the contents of this book on reversing diabetes Type 2. I explain how it begins with insulin toxicity …

Insulin spikes | Ali Diabetes

Posts about Insulin spikes written by Majid Ali MD. … Reversing Prediabetes and Diabetes With 3D Plan: Insulin-Wise and Insulin-Unwise Foods and Meals. Posted on July 24, 2017 by Majid … Video for majid ali diabetes insulin videos ▷ 5:57.

Insulin-Monitored Diabetes Reversal | Ali Diabetes

Sep 30, 2017 – Majid Ali, M.D. Yes, almond butter is an insulin-smart food. … List of Videos for Learning and Implementing Dr. Ali’s Insulin-Based Diabetes …

3D Insulin Protocol | Ali Diabetes

Posts about 3D Insulin Protocol written by Majid Ali MD. … Diabetes is a two-faced disease, one withinsulin toxicity and the other with insulin depletion: this diabetes duality in itself is most revealing. ….. https://vimeo.comMajid AliVideos.

Dr. Ali’s Insulin Reduction Protocol

Majid Ali, M.D. … For individuals with pre-diabetes with insulin toxicity but without high blood sugar levels, my … I present this subject at length in my book entitled “Dr. Ali’s Plan for Reversing Diabetes” and in a 40-minute video seminar that can …

Shortlink

Dr. Ali’s Breakfast Shakes

 

Optimal and Inappropriate Laboratory Testing For Assessing Insulin Homeostasis

Majid Ali, M.D.

Grievous Errors in Insulin Testing


 

What Is Optimal Laboratory Insulin Testing?

What Are Commonly Made Grievous Insulun Testing Errors?

 Optimal laboratory testing for assessing insulin homeostasis is to use tests that directly and specifically assess various aspects of insulin homeostasis. Inappropriate laboratory testing for assessing insulin homeostasis is to use tests that do not directly and specifically assess various aspects of insulin homeostasis.
 
Examples of optimal laboratory tests for insulin homeostasis are measurement of blood insulin concentration with fasting blood samples and timed samples obtained after a standard glucose challenge. Examples of inappropriate insulin tests are fasting blood glucose level, two-hours post-prandial blood glucose level, and A1c since these tests are test for glycemic status and not for assessing insulin homeostasis. 

Grievous Errors In Insulin Laboratory Tests
 
I recognize the following commonly made grievous errors in laboratory assessment of insulin homeostasis. Regrettably, these errors are deemed optimal standards for many doctors. 
 
1.   Blood insulin tests are done on randomly drawn blood tests (Results of such tests                              simply cannot be interpreted).
2.   The epidemic prevalences of hyperinsulinism of varying degrees are near-completely                     ignored in clinical medicine and insulin tests are simply not done (Table 2). 
3.   Tests for blood  sugar levels are done as substitutes for insulin tests. Glucose tests                            and others for glycemic status simply are not insulin tests.
4.   Laboratories use wholly inappropriate references ranges for blood insulin concentrations (See Table 2 for specifics). 
5.   Cut-off points for blood insulin concentrations determined with timed, post-glucose-                   challenge are not based on real insulin testing data.
6.   Insulin is the primary pro-weight gain and pro-obesity hormone, and yet insulin tests                 are done in weight loss and obesity programs. 
7.  Gestational diabetes is an insulin disorder before it becomes a glucose (sugar)                                 disorder. Insulin tests are not done for gestational diabetes.
8.  Insulin in excess is a potent the primary pro-weight gain and pro-obesity hormone,                       and yet insulin tests are done in weight loss and obesity programs. 
9. Insulin in excess is proinflammatory, pro-infections, pro-cancer, pro-premature aging,                 and pro-degenerative disorders and yet insulin tests are seldom, if ever, done by                 most doctors. 
10. Indeed, insulin in excess increases the risk of and fans the fires of all nearly chronic                  diseases 

Two Subtypes of Type 2 Diabetes: T2D Subtype A and T2D Subtype B
In 2014, I recognized the need to subtype Type 2 diabetes (T2D) into two T2D subtypes:
                              T2D subtype A
                               T2D subtype B
Diabetes is a two-faced disease, one with insulin toxicity and the other with insulin depletion: this diabetes duality in itself is most revealing. Below we present five sets of illustrative insulin and glucose profile taken from our original communication to make and illustrate our main points, which are presented and its full clinical implications considered in a separate chapter For the first five, ten or more years, the disease is characterized by rising blood sugar levels accompanied by increasing blood concentrations of insulin (hyperinsulinism aptly designated insulin toxicity). In the later years, T2D is characterized by rising blood sugar levels accompanied by falling insulin levels, this is the stage of insulin depletion (see Tables 1.1 and 1.2 for details).
Table 1. Insulin Homeostasis Categories in 506 Study Subjects Without Type 2 Diabetes
Insulin Category*
Percentage of Subgroup
Mean Peak Glucose  mg/dL
(mmol/mL)
Mean Peak Insulin (uIU/mL)
Exceptional Insulin Homeostasis.N 12**
1.7%
110.2     (6.12)
14.3
Optimal Insulin Homeostasis N =126
24.9 %
121.2     (6.73)
26.7
Hyperinsulinism, Mild                N =197
38.9 %
136.5   (7.58)
58.5
Hyperinsulinism,  Moderate       N =134
26.5 %
147.0    (8.16)
109.1
Hyperinsulinism,  Severe             N =  49
9.7 %
150.0    (8.33)
(less than time and a half higher) 
231.0
(nearly 17 times higher)
#   Correlation coefficient, r value, for means of peak glucose and insulin levels in the five insulin categories is 0.84.
*Criteria for classification: (1) Exceptional insulin homeostasis, a subgroup of optimal insulin homeostasis with fasting insulin concentration of <2 uIU/mL and mean peak insulin concentration of <20; (2) optimal insulin homeostasis, peak insulin <40 accompanied by unimpaired glucose tolerance; (3) mild
 


Table 2.  Insulin Reference Ranges  in uIU/mL of Six Laboratories in New York Metropolitan Area*
 Laboratory
 Fasting
 1 Hr
 2 Hr
 3 Hr
 Laboratory 1
1.9 – 23
8  –  112
 5 – 35
 Not Reported
 Laboratory 2
 2.6 – 24.9
 0.0  – 121.9
 0.0 – 163.5
 Not Reported
 Laboratory 3 
 2.6 – 24.9
 8  –  112
 5  –  55
 3  –  20
 Laboratory 4
 6  – 27
 20  –  120
 18  –  56
 8  –  22
 Laboratory  5
 00  – 30
 30  –  200
 40  – 300
 50  – 150
 Laboratory 6
 Does not include insulin ranges in the report. Instead it includes the following note: Insulin analogues may demonstrate non-linear cross-reactivity in this essay. Interpret results accordingly.**
*Upper and lower limits of laboratory reference ranges for blood insulin concentration determined following a Standard 75-gram glucose challenge.
**Personal communications with clinicians revealed that they do not find this laboratory note to be satisfactory in their clinical decision-making.

Grievous Errors in Insulin Testing

First Grievous Error: Believing That Diabetes (T2D) Is a Sugar (Glucose) Problem 
The first grievous error of considering insulin insufficiency as the cause of T2D has misled generations of doctors, leading to the mistreatment of hundreds of millions of people with prediabetes and T2D. In reality, hyperinsulinism predates T2D for five to ten or more years, although the study of insulin homeostasis is not deemed a standard of care for health preservation and disease prevention and/or control. Indeed, it is not taught in medical schools or on hospital wards, even where there are patients with suspected or diagnosed diabetes. The neglect of this core aspect of insulin dysregulation results in: (1) delayed diagnosis of T2D, and (2) as we document conclusively, the failure to detect and address long-established metabolic, inflammatory, immune, cardiovascular, and neurological consequences of insulin hyperinsulinism (Bahi-Buisson et al., 2008; Dandona, Aljada and Bandyopadhyay, 2004; IDFDA, 2016; Khan, Hull and Utzschneider, 2006; Shoelson, Lee and Goldfine, 2006; Shulman, 2014; Wellen and Hotamisligil, Shargill and Spiegelman,2005). Notable in this context is the recent documentation of hyperinsulinism in autism and pediatric dysautonomia (Ali, 2017a), which is discussed in chapter 6.
During the years of excess insulin – hyperinsulinism, or more appropriately insulin toxicity – widespread damage is inflicted in nearly all cell populations in the body. There is a profound irony here.  The very definitions of T1D and T2D lays bare the falsehood of the prevailing belief, the former being a state of near-complete absence of insulin in the blood while the latter for years is accompanied by raised blood insulin concentrations (as documented in Table 1.2). To add to the irony of this, consider the definition of insulin from the website of Merriam Webster Dictionary (March 15, 2017) reproduced verbatim here:
a protein pancreatic hormone secreted by the beta cells of the islets of Langerhans that is essential especially for the metabolism of carbohydrates and the regulation of glucose levels in the blood and that when insufficiently  produced results in diabetes mellitus …and that when insufficiently  produced [insulin] results in diabetes mellitus!
Consequently, it is not surprising that this utterly false notion of T2D caused by insulin insufficiency has become so deeply entrenched in public consciousness? The enduring belief of medical and nursing communities in this misleading dogma is of great concern. The key question is why has this definition not been previously challenged by the medical community?
To bring this grievous error into yet sharper focus, T1D is an acute-onset type disease usually occurring in children, characterized by near-complete absence of insulin-producing capacity of the pancreas gland. By contrast, T2D develops insidiously and, until recently, nearly always developed in adults. The blood insulin concentrations begin to fall after decades of insulin waste that occurs during the hyperinsulinism phase of the disease: this is what medical students learn in classrooms and on medical wards and  what nurses learn in nursing schools. Then the medical tragedy happens. Simple blood tests, for determining blood insulin concentrations to assess the state of insulin homeostasis of individual patients, is not considered a standard of care in any medical specialty or general practice. This disturbing notion of T2D being rooted in insulin insufficiency persists and so the hazards of insulin toxicity go unrecognized.

Second Grievous Error
Neglect of a Specific Quantitative and Modifier Marker
 The Third Grievous Error: Absurd Laboratory Insulin References Ranges
The third grievous error concerns laboratory reference ranges for blood insulin concentrations reported by most university hospital and nationwide commercial laboratories. Rather than guide clinicians interested in the study of insulin dysregulation in their patients, clinical pathologists and laboratory professionals have for decades compounded the problem of neglected hyperinsulinism. Table 1.3 displays wide variations in the lower and upper limits in the reference ranges for fasting and post-glucose challenge blood insulin concentrations employed by six major laboratories in the New York City metropolitan area. The variation in insulin reference ranges invariable invites skepticism, with photographs of actual laboratory reports on the web (www.alidiabetes.org). Note that laboratory 1 reports a range of 5-35 for 2-hour blood insulin level while laboratory 5 reports of range of 40-300 for the sample blood sample: while laboratory 1 reports a range of 5-35 for 2-hour blood insulin level. Further, laboratory 5 reports of range of 40-300 for the sample blood sample, while laboratory 2 reports a range of 0.0 to 121.9 and laboratory 4 reports 20-120 for the same blood sample. It is difficult to imagine a parallel for this level of absurdity in the entire field of laboratory medicine.

Cut-off Points for Optimal Insulin Homeostasis and Degrees of Hyperinsulinism
Our selection of the peak insulin value of <40 mIU/mL as the cut-off point for optimal insulin homeostasis in our survey of prevalence of hyperinsulinism in New York (see Table 1.1), was based on a preliminary review of the first 50 sets of insulin and glucose profiles (Ali et al., 2017a). We opted for cut-off points for hyperinsulinism stratification based on doubling of the levels (to <80, <160, and >160 uIU/mL for mild, moderate, and severe hyperinsulinism) with two considerations: (1) are these cut-off points appropriate for this study, and (2) do they provide a frame of reference for future investigations of diverse aspects of insulin homeostasis and hyperinsulinism-to-T2D progression? There are a number of other issues that need to be considered in this context: (1) what constitutes optimal insulin homeostasis, (2) what should the insulin cut-off point be, as there is no agreement within the relevant literature, (3) no adverse effects of low insulin levels when accompanied by unimpaired glucose tolerance have been reported, and (4) Hyperinsulinism and the metabolic syndrome are commonly spoken in the same breath,  explicitly or implicitly referring to them as the two faces of the same coin. However, there is a crucial difference between the two, the peak insulin level and other features of three-hour insulin and glucose profiles provide clinicians with  specific and quantitative cut-off  points for detecting and stratifying hyperinsulinism but no such criteria have been established for the metabolic syndrome. In addition, three-hour insulin and glucose profiles shed light on other aspects of glycemic status and insulin homeostasis, some of which are presented later in this chapter.
A subgroup of twelve participants was designated ‘exceptional insulin homeostasis’ for two reasons: (1) they showed an extremely low fasting insulin value of <2 uIU/mL (mean 14.3 uIU/mL) and peak insulin concentrations <20 uIU/mL accompanied by unimpaired glucose tolerance, and (2) ten of the twelve had no family history of diabetes (parents, siblings, grandparents, children, uncles or aunts), while the mother of the eleventh subject developed T2D in the closing months of her life at age 74 and both parents of the twelfth subject had T2D. This subgroup appears to reflect ideal metabolic efficiency of insulin in the larger evolutionary context.

Shifting Focus from Glucose Testing to Insulin Testing
As reported in the preface, the much higher rate of hyperinsulinism observed in New York’s general population compared to rates of T2D in India (Kaveeshwar and Cornwell, 2014) and China (Xu et al., 2013), provides strong support for the view that there is a need to shift focus from glucose testing to insulin testing for stemming global tides of hyperinsulinism and T2D. A crucial point in this context is that the data published in the Indian and Chinese studies was derived from glucose testing, whereas our insulin database was derived exclusively from direct insulin testing, with measurements of post-glucose challenge blood insulin concentrations with sequential and timed blood samples.
Here we point out that the insulin and glucose profiles presented in this and other chapters shed light on the full spectra of insulin homeostasis, hyperinsulinism and related patterns of insulin dysfunction, for example insulin spikes followed by hypoglycemic episodes which create hunger for foods that create yet more sugar spike. Therefore the insulin and glucose profiles presented in Tables 1.4-1.8 in this (and numerous in other chapters) require that the data be considered in light of the clinical context as well as looking through the kaleidoscopic prisms of molecular biology of oxygen Ali, 2000, 2002, 2004a, 005a, 2007, 2009a, 2011), oxygen model of hyperinsulinism (Ali, 2014a) and oxygen model of T2D (Ali, 2001). As for co-morbidities of the hyperinsulinism-T2D continuum (metabolic, inflammatory, immune, infectious, cardiovascular, neurological, developmental, gut-microbiota-related, differentiative, and degenerative), we do not recognize any  inconsistencies between our observations and inferences and those of earlier workers (Nath, Heemels and Anson, 2006; Nichols, 2012; Patti et al., 2003; Saltiel and Kahn, 2001; Scherer, 2005; Stanley, 2016; Turnbaugh, 20

 


Table 3. Insulin Homeostasis Categories in 178 Study Subjects With Type 2 Diabetes
Insulin Category
Percentage of Subgroup
Mean Peak Glucose, mg/dL
(mmol/mL)
Mean Peak Insulin (uIU/mL)
Diabetic Hyperinsulinism, Mild              N =  53
29.0%
252.0   (14.00)
55.4
Diabetic Hyperinsulinism, Moderate    N =  42
24.0%
242.1   (13.45)
112.4
Diabetic Hyperinsulinism, Severe          N =  24
13.9%
224.6   (12.47)
298.0
Diabetic  Insulin Deficit                             N =  59
33.1%
294.0    (16.33)
22.9
Illustrative Case Studies of Insulin Responses to Glucose Challenge
Tables 4 to 8 present five illustrative sets of insulin and glucose profiles with brief clinical notes. The insulin profiles in Tables 4 and 8  represent the two extremes of insulin peaks (18 uIU/mL and 718.2 uIU/mL) encountered in this survey. The first of the two profiles (Table 4) is reflective of ideal metabolic efficiency of insulin in a larger evolutionary perspective of energy economy in the body. Notable findings here are: (1) a very low fasting insulin level of <2 uIU/mL reflecting efficient insulin conservation during the fasting state; (2) low insulin peak value (18 uIU/mL) indicating high insulin efficiency following a substantial glucose challenge; and (3) a very low insulin level in the 3-hour sample (<2 uIU/mL) reflects optimal beta cell response to glucose level falling below the fasting level.
 
Table 4. Example of Insulin and Glucose Profiles In Exceptional Insulin Homeostasis Category*
 
Fasting
½ Hr
1 Hr
2 Hr
3 Hr
Insulin uIU/mL
<2
18
14
4
<2
Glucose mg/mL  (mmol/L)
77     (4.27)
168   (9.33)
109      (6.05)
74       (4.11)
59    (2.88)
*The Patient,  A  60-Yr-Old 5’ 7” Man Weighing 138 lbs. Presented for a Wellness Assessment. He Was Considered to be in Excellent Health By Clinical and Laboratory Evaluation Criteria.
Table 5.  Severe Hyperinsulinemia in A Subject With Previously Undiagnosed Type 2 Diabetes*
 
Fasting
½ Hr
1 Hr
2 Hr
3 Hr
Insulin uIU/mL
23.8
19.3
36.9
114.7
75.2
Glucose mg/mL  (mmol/L)
112     (6.21)
158   (8.77)
214      (11.76)
241    (13.38)
129   (7.16)
* The Patient,  A 64-Yr-Old 5’ 4” Woman Weighing 164 lbs. Presented With Hypothyroidism, History of Coronary Artery Stent Insertions, Fatty Liver, Memory Concerns And Without Previous Diagnosis of Type 2 Diabetes.
Table 6. Hyperinsulinism 18 Years After the Diagnosis of Type 2 Diabetes*
Fasting
½ Hr
1Hr
2Hr
3Hr
Insulin uIU/mL
  12.9
27.2
29.2
36.2
25.4
Glucose mg/mL  (mmol/L)
128      (7.10)
224   (12.43)
278    (15.42)
297    (16.48)
249     (13.81)
*The Patient,  A 74-Yr-Old 5’ 6” Woman Weighing 155 Lbs. Presented With Bronchiectasis, Rheumatoid Arthritis, Prehypertension, and Inhalant Allergy.
Table 7. Brisk Insulin Response With A “Flat” Glucose Tolerance Profile*
Fasting
½ Hr
1Hr
2Hr
3Hr
Insulin uIU/mL
3
23
22
8
<2
Glucose mg/mL  (mmol/L)
72      (3.39)
44     (2.44)
63    (3.49)
58     (3.21)
65   (3.90)
*The Patient,  A 47-Yr.Old  5’ 5” Woman Weighing 170 Lbs. Presented With Polyarthralgia, Recurrent Sinusitis, and Fatigue.
Table 8. Severe Hyperinsulinism In A 13-Yr-Old Girl With Lupus Erythematosus*
Fasting
½  Hr
1Hr
2Hr
3Hr
Insulin uIU/mL
27.9
362.5
424.0
718.2
571.7
Glucose mg/mL  (mmol/L)
      70   (3.88)
  140     (7.77)
   157     (8.71)
   150    (8.33)
   111   (6.16)
Insulin and Glucose Profiles Obtained After Four Months of Robust Integrative Therapies
Insulin uIU/mL
7.2
125.1
238.5
208.0
132.0
Glucose mg/mL  (mmol/L)
81     (4.49)
154   (8.54)
181     (10.04)
130     (7.21)
97      (5.38)
*The Patient,  A 13-Yr-Old Girl With a History of Three Hospitalizations In One Year for Systemic Lupus Erythematosus, Recurrent Pneumonia, Thrombocytopenia, and Severe Optic Neuritis Resulting In Complete Loss of Vision In Right Eye. The Peak Insulin Fell from 718 to 238.5 In Four Months of Robust Integrative Treatment.
 

Hyperinsulinism Associated With Breast and Prostate Cancer

Majid Ali, M.D.

Published in the Journal Townsend Letter (2017;409:66-69

(August 2017)


 

Hyperinsulinism fans the fire of cancer. In this article, I present case studies to show diet and integrative therapies can restore insulin homeostasis and, thereby:

  1. Reduce the risk of prostate and breast cancer growth.
  2. Improve results in the treatments of these cancer.

Two Dimensions of Insulin Dysregulation

 

An Essay Competition Article
By
Aliza Durrani
Age 13
Cherry Hill, New Jersey
7/19/17

 

In the article written by Doctor Majid Ali I have learned that Type 1 diabetes can’t get reversed while Type 2 can. This happens because Type 1 DM produces none to very little insulin while type 2  DM produces either a lot of insulin which does not work due to resistance or produce little to none insulin because their pancreas is failing after not being treated. In the article I agree with the author when he says we should be checking insulin level early on. In routine practice I am shocked that we don’t check insulin level and we only check glucose level. As a 13 year old that is a no brainer to me. If we check insulin level early on we can reverse the type 2 diabetes and since we only check glucose level this can cause people to go years to decades without being treated, resulting by the pancreas to burn out.


Insulin Diet, Insulin Detox, and Insulin Dysox

In the article I have learned that the glucose and insulin levels should be going down at the 2 hour mark in someone’s body who is not diabetic, but in the table the information is reported for a 75 year old women and it shows the glucose level and insulin level escalating each hour and not going down at the 2-3 hour mark, which means the person is diabetic and is insulin resistant. After the patient had completed a successful 3D protocol including diet, detox,and dysoxic comorbidities the last test taken on the graph was April 14, 2015 the test results appear to be normal because the levels were controlled and went down at the 2-3 hour mark resulting to her diabetes to be reversed. Also I have gained knowledge about Hyperinsulinism, it causes inflammation to organs, which can cause cancer, strokes,heart attack, etc. Hyperinsulinism is the pancreatic response to increased energy requirement to repair the injured tissue.


Two Dimensions of Insulin Dysregulation

Lastly I have learned that insulin dysregulation has two dimensions the first one is pathophysiology of hyperinsulinism(predates Type 2 DM and is not accompanied by glycemic),and the last one is dimension of T2D(accompanied by hyperglycemia). This article has given me a lot of knowledge and gives me a new look into the medical field. Studies like this will help the future to find cures for other medical issues.

Reversing Diabetes: Four Goals for the Patient, Four for the Physician

Majid Ali, M.D.

The Path Away From Diabetes


Four Steps for the Patient

  1.                                  Love

  2.                                 Oxygen

  3.                                 Prayer

  4.                                 Insulin


 

Love provides the purpose of reversing diabetes.

Prayer provides the path to that purpose.

Oxygen orchestrates the bodily functions that prevent and reverse diabetes.

Insulin is the master energy hormone for preventing and reversing diabetes.


 

The Path

The Path to Healing Is the Path to Peace.

The Path to Peace Is the Path to the Spiritual.

The Path to the Spiritual Is the Path Away From the Self.

The Path Away from the Self Is the Path to One’s Own Divinity, to Oneness With Divinity.

The Path to That Oneness Is the Path to Insulin Solutions, and Freedom from Diabetes and Its Suffering.


 

Four Steps for the Physician

My top four goals for reversing diabetes and clearing insulin toxicity (hyperinsulinism) are the same as they for all other chronic diseases. Specifically, they are:

                                      1. Oxygen signaling  

                                 2. Gut ecology  

                        3. Insulin signaling 

 4. Truth and best effort


 

For my patients on the Path Away From Diabetes, I 

  • Promise no results,
  • Promise total honesty, and 
  • Promise best effort.

 

An Important Link for the Path

http://www.insulininstitute.org/dr__a__insulin_protocol_a.htm

Other important links for knowing the path, please go to my video library near the end.  


Plant Remedies and Supplements

for Reversing Hyperinsulinism (Prediabetes) and Diabetes

Phytofactors (Plant Remedies) 

  1. Aloe Vera
  2. Bitter melon
  3. Cinnamon
  4. Fenugreek
  5. Flaxseed
  6. Garlic
  7. Ginseng (Panax ginseng)
  8. Gymnema sylvestre
  9. Huckelberry
  10. Neem
  11. Nopal (prickly pear cactus)

Nutritional Supplements

  1. Coenzyme Q10
  2. Chromium
  3. Lipoic acid (alpha lipoic)
  4. Magnesium, potassium, Taurine
  5. Cinnamon
  6. Chromium
  7. Vanadyl sulfate
  8. Multivitamin
  9. Multimineral

Top Priorities for Nutrients and Plant-Based Remedies 

1. Magnesium, Potassium, and Taurine

2. Calcium-magnesium

3  Multiminerals (selenium, chromium, molybdenum, vanadium, manganese),

4. Multivitamins

5. Plant-based remedies

 


Magnesium, Potassium, and Taurine

I prescribe magnesium, potassium, and taurine for every patients when my goals are:

  1. To reverse diabetes
  2. To clear insulin toxicity of hyperinsulinism
  3.  To prevent complications of Type 2 diabetes
  4. To  alter the course of other types of diabetes.

 

Magnesium, Do Not Forget To Take It, Please! Majid Ali MD on Vimeo

https://vimeo.com › Majid Ali › Videos

Magnesium, Do Not Forget To Take It, Please! Majid Ali MD. 3 years ago More.Majid AliPRO. Follow. 0 …

How Much Magnesium Do I Take Majid Ali MD on Vimeo

https://vimeo.com › Majid Ali › Videos

How Much Magnesium Do I Take Majid Ali MD. from Majid AliPRO 3 years ago. Follow. 0 0. Download Share …

Magnesium and the Heart – Dr. Ali’s Virtual Medical Library

drali1.org/magnesium_and_heart.htm

Magnesium and the Heart. Majid Ali, M.D.. A large body of data showing a relationship between low dietary intake of magnesium and the incidence of cardiac …

Healing Foods by Majid Ali, MD Note: The … Soybean: is an excellent source of minerals such as magnesium, calcium, molybdenum and others. It is rich in …Magnesium and the Heart – Dr. Ali’s Virtual Medical Library

drali1.org/magnesium_and_heart.htm

Magnesium and the Heart. Majid Ali, M.D.. A large body of data showing a relationship between low dietary intake of magnesium and the incidence of cardiac …


 

Magnesium Content of Foods | The Ali Academy Community

Jun 20, 2014 – Magnesium, The Miracle Mineral Majid Ali, M.D. Magnesium is an … I liberally prescribepotassium and magnesium supplementation for all my …


Your Kidney’s New Year Resolution | Renal Health | The Ali Academy …

Dec 29, 2014 – The kidneys help keep the right amount of potassium in the body. … Potassium, Sodium, and Neurotransmission Majid Ali MD – …

Dr. Ali Expains How Minerals Beat Fatigue | The Ali Academy …

Jun 1, 2014 – Minerals: Your Body’s Energy Sparks Majid Ali, M.D. There are two types of … Potassium, Sodium, and Neurotransmission Majid Ali MD – …

How Much Potassium Do I Take Majid Ali MD on Vimeo

https://vimeo.com › Majid Ali › Videos

Join · Log in · Host videos · Compare plans · Professionals · Businesses · Video lovers · Video School …

Dr. Ali’s Spicy Potassium Lemonade – Majid Ali, MD on Vimeo

https://vimeo.com › Majid Ali › Videos

This is “Dr. Ali’s Spicy Potassium Lemonade – Majid Ali, MD” by Majid Ali on Vimeo, the home for high …

Dr. Ali’s Potassium Lemonade – Majid Ali, MD on Vimeo

https://vimeo.com › Majid Ali › Videos
Aug 7, 2014

This is “Dr. Ali’s Potassium Lemonade – Majid Ali, MD” by Majid Ali on Vimeo, the home for high quality …


Taurine 

A free course on Liver Health NOW | Majid Ali MD | The Ali Academy …

Nov 28, 2014 – Inflammation and Liver – Majid Ali, MD ….. methylsulfonylmethane (MSM, 750 to 1,500 mg); taurine (750 to 1,500 mg); and antioxidant vitamins.

Taurine – Majid Ali, MD on Vimeo

https://vimeo.com › Majid Ali › Videos
Apr 23, 2014

Professor Majid Ali shares information about “Taurine

Ali Healing Community

Majid Ali, M.D Misinformation about the science of health, eating depleted … Why do I commonly prescribe magnesium, potassium, and taurine for most of my …

Majid Ali MD | – alihealing.org

Read all of the posts by Majid Ali MD on. … Why do I commonly prescribe magnesium, potassium, andtaurine for most of my patients with chronic disease in my …

Integrated Magnesium Therapy – Who Is Dr. Ali?

Aug 3, 2017 – Majid Ali, M.D. I liberally prescribe integrated magnesium … magnesium prescriptions nearly always include potassium, calcium, and taurine.

 


 


Vitamin B6 for Preventing and Reversing Gestational Diabetes

Vitamin B 6 in its Pyridoxal-5-phosphate (P5P) form can be rightfully considered a nutrient of choice for preventing and reversing gestational diabetes. Simply stated, this is how it works:

  1. Xanthourenic acid (XA) is a metabolite of tryptophan(which is used to produced melatonin and serotonin)
  2. Women who are more vulnerable to gestational diabetes seem to have altered enzymatic function so tryptophan is readily turned into xanthourenic but not so readily into melatonin and serotonin.
  3. Excess xanthourenic acid binds with insulin and blocks metabolic functions of insulin in lowering blood glucose level setting the stage for gestational diabetes..
  4. Women who are more vulnerable to gestational diabetes seem to have altered enzymatic function so tryptophan is readily turned into xanthourenic but not so readily into melatonin and serotonin.
  5. Very high estrogen levels during pregnancy also seem to play a role.

 

 

Top Priority Nutrients for Reversing Diabetes By Clearing Insulin Toxicity

Majid Ali, M.D.   Top Priorities for Nutrients and Plant-Based Remedies  1. Magnesium, Potassium, and Taurine 2. Calcium-magnesium 3  Multiminerals (selenium, chromium, molybdenum, vanadium, manganese, 4. Multivitamin   …

What Is Empirical Healing?

Majid Ali, M.D. All healing is energy healing. Since humans began to look for ways to deal with illness, it sought therapies that worked and were safe. In earlier times, safe and effective therapies were not discarded ju…

Dr. Ali’s Insulin Library

Majid Ali, M.D. My Oxygen Thinking Has Given Me Insights About the Roles of Insulin in Health and Disease which Robustly Challenge the Prevailing Notions of Insulin, Insulin Resistance, and Hyperinsulinism.    Large Clai…

Not Moving Away From Diabetes Is Moving Towards It

  Majid Ali, M.D. Insulin toxicity and diabetes have eclipsed All Chronic Diseases Worldwide. I am grateful to my Patients (My Truest Teachers) Who Helped Me Recognize This Disturbing Reality.    Insulin Essentials Insul…

Insulin Essentials

Majid Ali, M.D. Very little of What I Learned About Diabetes In Medical School Has Been Validated by My Patients, My True Teachers.   Insulin Essentials Insulin is the master energy hormone of the body, for energy genera…

Reversing Diabetes D3 – Part A: The Diet Plan

Majid Ali, M.D. A Simplified Yet Effective Choices in-the-Kitchen Part of Reversing Diabetes D3 Plan for Preventing and Reversing Diabetes Based on Authentic Science and Philosophy of Holism    Reversing  Diabetes D3    …

Diabetes and Insulin Library

Majid Ali, M.D. A Comprehensive Library of the Science and Philosophy of Holism For Preventing and Reversing Diabetes    Diabetes Reversal With Insulin Detox | Ali Diabetes https://alidiabetes.org/category/diabetes-rever…
Majid Ali, M.D. Alzheimer’s Disease Before the Supreme Court of Science – 2017     Alzheimer’s Before the Supreme Court of Science – 2017 I have subscribed and read the journal Nature over 25 for years. It is a journal o…
Three Stages of Diabetes
Majid Ali, M.D. Do You Know Which One of the Three Stages of Diabetes You Are In? Why That Is Important?   You can Learn This Only With Blood Insulin Test. Diabetes In Hyperinsulinism Stage With High Blood In…
  • Should A1c Tests Be Used for Screening for Diabetes? No.

Majid Ali, M.D. Insulin and Glucose Profiles of Reversing Diabetes D3 Series     The blood A1c test is an excellent test for monitoring the results of diabetes treatment, but it is not reliable for screening for diabetes…

 

 

 

 

 

 

 

Not Moving Away From Diabetes Is Moving Towards It

 

Majid Ali, M.D.

Insulin toxicity and diabetes have eclipsed All Chronic Diseases Worldwide. I am grateful to my Patients (My Truest Teachers) Who Helped Me Recognize This Disturbing Reality. 


 

Insulin Essentials

  1. Insulin is the master energy hormone of the body, for energy generation as well as energy expenditure.
  2. The energy demands of chronically-injured cells increase because repair of injured tissues needs more energy.
  3. Increased demands for cellular repair energy can be met only with increased supply of fuel (glucose) for producing more cellular energy.
  4. Higher demands for glucose require higher insulin activity.
  5. The validity of these statements can be tested only with direct blood insulin tests, not by doing blood tests for glucose (fasting blood glucose, A1c test, two-hour post-prandial blood sugar, or three-hour glucose tolerance test after a glucose load.
  6. other forms of sugar.
  7. Anyone can test the validity of the above statement with blood insulin tests.

 

What My Professors Did Not Tell Me About Insulin Essentials

  1. Newborn babies with birth weight larger than eight pounds are insulin toxic.
  2. Mothers of babies with birth weight larger than eight pounds are insulin toxic.
  3. Expecting moms with gestational diabetes are insulin-toxic and will remain so after delivering their babies for variable periods of time.
  4. Boys with widespread persistent acne are insulin-toxic.
  5.  Young girls with polycystic ovarian cystic syndrome are insulin-toxic.
  6. Nearly all obese children are insulin-toxic.
  7. Children and adults with fatty liver and steatosis are insulin-toxic.
  8. Most patients with pulmonary fibrosis, bronchiectasis, and active tuberculosis are insulin-toxic. 
  9. Most individuals with psoriasis and sarcoidosis are insulin-toxic.
  10. Most individuals with chronic autoimmune disorders (rheumatoid arthritis, lupus, scleroderma, and others) are insulin-toxic.
  11. Most patients with chronic renal failure are insulin-toxic.
  12. Most individuals with memory loss, dementia, Alzheimer’s disease, and diverse chronic diseases of the brain are insulin-toxic.
  13. Most individuals with cancer are insulin-toxic.
  14. Nearly all people become insulin-toxic after receiving chemotherapy.

 

Dr. Ali’s Insulin Library


Community Texts : Free Books : Free Texts : Download & Streaming …

Majid Ali, M.D. Why I Don’t Recommend Skim Milk Products. – -. by Majid Ali, M.D.. texts … 20 20. 4. IJGMP Metabolic Syndrome. Aug 20, 2016 08/16. by IASET …

« Older Entries