Category Archives: Ali’s Fermentation Course

Is Diabetes A Sugar Problem? No.

Majid Ali, M.D.

Suite 3 C, 344 Prospect Avenue

Hackensack, New Jersey 07601



Is diabetes mellitus (Type 2 Diabetes) a sugar problem? No. The abnormalities of blood sugar seen in diabetes are the consequences of the derangements of cellular energetics and toxicity that collectively create what is commonly called diabetes. Is diabetes an insulin problem? No. The abnormalities of insulin functions are the consequences of plasticized (chemicalized) and hardened cell membranes that immobilize the insulin receptors embedded in them. Is diabetes a problem of blood vessels that causes blindness, kidney failure, stroke, heart attacks, and neuropathy? No. The abnormalities of blood vessels are the consequences of oxidizing and deoxygenizing influences in diabetes.

In this column, I marshal evidence for my view that the state of insulin resistance should be regarded as a “hardened cell membrane state.” The so-called metabolic syndrome should be visualized as a “gummed-up matrix state.” Prediabetes should be seen as a “mitochondrial dysfunction state.” The strategies for the prevention and reversal of diabetes yield better long-term clinical results if diabetes is recognized as a “dysfunction oxygen signaling,” or dysox, state.

In type 1 diabetes, insulin itself becomes a potent autoantigen and initiates autoimmune injury to pancreatic islet cells.1-3 I will show how this recently documented role of insulin in the pathogenesis of diabetes fits in the dysox model of diabetes presented here. In type 2 diabetes, insulin cannot function – insulin resistance, in the common jargon – and hyperinsulinemia develops, which triggers and amplifies the inflammatory response.4-6 In all types of diabetes, the endothelial cells produce nitric oxide and other bioactive factors in abnormal quantities and proportions.7,8 Diabetes causes neuropathy, retinopathy, nephropathy, dementia, stroke, and heart attacks. I will describe how those complications of diabetes can be better understood when the problems are seen through the prism of oxygen signaling.


Clinical, Epidemiologic, and Experimental Evidence Links Obesity With Insulin Toxicity

The link is supported by known metabolic roles of nonesterified fatty acids (NEFAs) and altered paracrine and endocrine functions of fat cells (adipocytes) in the energy economy of the body. For example, in a healthy state, NEFAs serve as substrates for adenosine triphosphate (ATP) generation. In obesity, these fatty acids are retained in excess in biomembranes of all cell populations of the body and within adipocytes. NEFAs, along with trans fats and oxidized lipids, then “harden” the cell membranes to clamp down on insulin receptors – rusting and impacting the crank, so to speak – to cause insulin resistance.12 Those lipids also “gum up” the matrix, blocking molecular cross-talk there. Eventually, those elements, along with other toxins, uncouple respiration from oxidative phosphorylation and impede mitochondrial electron transfer events.


In obesity, output of fattening hormones in adipocytes (fat cells) is chaotic in the ways in which it further increases cellular fat build-up and sets the stage for the development of diabetes.13,14 However, the obesity/diabetes link does not prevail in all populations of the world. For instance, in India, there is also an epidemic of low body-weight (LBW) diabetes15 – a phenomenon that clearly points to the existence of environmental factors unrelated to obesity that are involved in the pathogenicity of diabetes, and supports the dysox model of diabetes.

A growing number of free radicals, transcription factors, enzymes, and proteins has been – and continues to be – implicated in the pathogenesis of diabetes, including:
· nitric oxide16,17
· inducible nitric oxide synthase (iNOS)18
· mitochondrial uncoupling proteins (UCPs)19-21
· proinflammatory cytokines22-24
· resistin25,26
· leptin27,28
· adipokines29
· adiponectin30
· tumor necrosis factor-alpha (TNF-a)31
· peroxisome proliferator-activated receptor gamma (PPARgamma)32-34
· nuclear respiratory factor-1 (NRF-1)35
· suppression of cytokine signaling (SOCS) proteins36
· retinol-binding protein-4 (RBP4)37
· antibodies against glutamic acid decarboxylase38
· prothrombotic species, including fibrinogen, von Willebrand factor, and plasminogen activator inhibitor (PAI-1), adipsin (complement D), and acylation-stimulating protein (ASP) 39-42
· heat shock protein 60, voltage-dependent anion channel 1 (VDAC-1), and Grp7543
· hypercoagulable platelets44

Oxygen, Diabetes, Insulin References 

1.Nakayama M, Norio Abiru N, Moriyama H, et al. Prime role for an insulin epitope in the development of type 1 diabetes in NOD mice.
Nature. 2005;435,220-223.
2.Kent SC, Chen Y, Bregoli L, et al. Expanded T cells from pancreatic lymph nodes of type 1 diabetic subjects recognize an insulin epitope.
Nature. 2005;435:224-228.
3.von Herrath M. Insulin trigger for diabetes.
Nature. 2005;435:151-152.
4.Eisenbarth GS, et al. Insulin autoimmunity: Prediction/precipitation/prevention type 1A diabetes.
Autoimmun. Rev. 2002;1:139-145.
5.Todd JA, Bell JI, McDevitt HO. HLA antigens and insulin-dependent diabetes.
Nature. 1988;333,710-712.
6.Ali M. Hypothesis: obesity is adipomyocytic dysoxygenosis.
J Integrative Medicine. 2004;9:19-38.
7.Wellen KE, Hotamisligil GS. Inflammation, stress, and diabetes.
J. Clin. Invest. 2005;115:1111–1119.
8.Shoelson SE, Lee J, Goldfine AB. Inflammation and insulin resistance.
J. Clin. Invest. 2006;116:1793–1801.
9.World Health Organization Consultation on Obesity 1–253 (World Health Organization, Geneva, 2000).
10.Wild S, Roglic G, Green A, et al. Global prevalence of diabetes: Estimates for the year 2000 and projections for 2030.
Diabetes Care. 2004;27:1047–1053.
11.Hedley AA. Prevalence of overweight and obesity among US children, adolescents, and adults, 1999–2002.
JAMA. 2004;291:2847–2850.
12.Leung, et al. Prolonged increase of plasma non-esterified fatty acids fully abolishes the stimulatory effect of 24 hours of moderate hyperglycaemia on insulin sensitivity and pancreatic beta-cell function in obese men.
Diabetologia. 2004;247:204–213.
13.Rosen ED, Spiegelman BM. Adipocytes as regulators of energy balance and glucose homeostasis.
Nature. 2006;444:847-853.
14.Nath D, Heemels M-T, Lesley Anson L Obesity and diabetes.
Nature. 2006;444, 839.
15.Das S. Identity of Lean-NIDDM: Clinical, metabolic and hormonal status. In: Kochupillai N, ed.
Advances in Endocrinology, Metabolism, and Diabetes. Vol. 2. Delhi, India: Macmillian; 1994:42-53.
16.Farmer SR. Transcriptional control of adipocyte formation.
Cell Metab. 2006;4:263–273.
17.Trayhurn P. Endocrine and signalling role of adipose tissue: New perspectives on fat.
Acta Physiol. Scand. 2005;184: 285–293.
18.Perreault M, Marette A. Targeted disruption of inducible nitric oxide synthase protects against obesity-linked insulin resistance in muscle.
Nature Med. 2001;7:1138–1143.
19.Suh YH, Kim SY, Lee H, et al. Overexpression of short heterodimer partner recovers impaired glucose-stimulated insulin secretion of pancreatic beta-cells overexpressing UCP2.
J Endocrinol. 2004;183:133-44.
20.Ceddia1 RB, William WN, FB, et al. Leptin stimulates uncoupling protein-2 mRNA expression and Krebs cycle activity and inhibits lipid synthesis in isolated rat white adipocytes.
Eur. J. Biochem. 2000;267:5952-5958.
21.Enerback S et al. Mice lacking mitochondrial uncoupling protein are cold-sensitive but not obese.
Nature. 1997;387:90–94.
22.Xu H. Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance.
J. Clin. Invest. 2003;112:1821–1830.
23.Shoelson, SE, Lee J. Goldfine AB. Inflammation and insulin resistance.
J. Clin. Invest. 2006;116: 1793–1801.
24.Murphy KG, Bloom SR. Gut hormones and the regulation of energy homeostasis.
Nature. 2006;444:854-859.
25.Stepphan CM, Bailey ST, Bhat S, et al. The hormone resistin links obesity to diabetes.
Nature. 2001:409;307-312.
26.Berti L, Kellerer M, Capp E, et al. Leptin stimulates glucose transport and glycogen synthesis is in C2C12 myotubes: Evidence for a P3-kinase mediated effect.
27.Minokoshi Y et al. Leptin stimulates fatty-acid oxidation by activating AMP-activated protein kinase.
Nature. 2002; 415: 339–343.
28.Farooqi IS, et al. Beneficial effects of leptin on obesity, T cell hyporesponsiveness, and neuroendocrine/metabolic dysfunction of human congenital leptin deficiency.
J. Clin. Invest. 2002;110:1093–1103.
29.Shimomura I, Hammer RE, Ikemoto S, et al. Leptin reverses insulin resistance and diabetes mellitus in mice with congenital lipodystrophy.
Nature. 1999;401:73–76.
30.Fain JN, Madan AK, Hiler ML, et al. Comparison of the release of adipokines by adipose tissue, adipose tissue matrix, and adipocytes from visceral and subcutaneous abdominal adipose tissues of obese humans.
Endocrinology. 2004;145:2273–2282.
31.Scherer PE. Adipose tissue: From lipid storage compartment to endocrine organ.
Diabetes. 2006;55:1537–1545.
32.Atherton HJ, Bailey NJ, Zhang W, et al. A combined 1H-NMR spectroscopy- and mass spectrometry-based metabolomic study of the PPAR-alpha null mutant mouse defines profound systemic changes in metabolism linked to the metabolic syndrome.
Physiol Genomics. 2006;27:178-186.
33.Kadowaki T et al. Adiponectin and adiponectin receptors in insulin resistance, diabetes, and the metabolic syndrome.
J. Clin. Invest. 2006;116:1784–1792.
34.Farmer SR. Transcriptional control of adipocyte formation.
Cell Metab. 2006;4:263–273.
35.Yang Q, et al. Serum retinol binding protein 4 contributes to insulin resistance in obesity and type 2 diabetes.
Nature. 2005;436:356–362.
36.Mooney RA, et al. Suppressors of cytokine signaling-1 and -6 associate with and inhibit the insulin receptor. A potential mechanism for cytokine-mediated insulin resistance.
J. Biol. Chem. 2001;276:25889–25893.
37.Patti ME, Butte AJ, Crunkhorn S, et al. Coordinated reduction of genes of oxidative metabolism in humans with insulin resistance and diabetes: Potential role of PGC1 and NRF1.
Proc Natl Acad Sci U S A. 2003;100:8466-8471.
38.von Boehmer H, Sarukhan A. DAG, a single autoantigen for diabetes.
Science. 1999;284:1135-1136.
39. Van Gaal LF, Mertens IL, De Block CE. Mechanisms linking obesity with cardiovascular disease.
Nature. 2006;444:875-880.
40.Matsuzawa Y. The metabolic syndrome and adipocytokines.
FEBS Lett. 2006;580:2917–2921.
41.Konstantinides S, Schafer K, Koschnick S, et al. Leptin-dependent platelet aggregation and arterial thrombosis suggests a mechanism for atherothrombotic disease in obesity.
J. Clin. Invest. 2001;108:1533–1540.
42.Bernal-Mizrachi E, Wen W, Stahlhut S, et al. Islet cell expression of constitutively active Akt1/PKB induces striking hypertrophy, hyperplasia, and hyperinsulinemia.
J. Clin. Invest. 2001;108:1631–1638.
43.Turko IV, Murad F. Quantitative protein profiling in heart mitochondria from diabetic rats.
J Biol Chem. 2003;278(37):35844-35849.
44.Lillioja S, Mott DM, Spraul M, et al. Insulin resistance and insulin secretory dysfunction as precursors of non-insulin-dependant diabetes mellitus: Prospective studies of Pima Indians.
N Engl J Med. 1993;329:1988-1992.
45.Sakaue M, Fuke Y, Katsuyama T, et al. Austronesian-speaking people in Papua New Guinea have susceptibility to obesity and type 2 diabetes.
Diabetes Care. 2003 26: 955-956.
46.Katulanda P, Sheriff MH, Matthews DR. The diabetes epidemic in Sri Lanka – a growing problem.
Ceylon Med J. 2006;51:26-28.
47.Landau BR, Chandramouli V, Schumann WC, et al. Estimates of Krebs cycle activity and contributions of gluconeogenesis to hepatic glucose production in fasting healthy subjects and IDDM patients.
Diabetologia. 1995;38:831-838.
48.Tian J, Zekzer D, Lu Y, et. al. B cells are crucial for determinant spreading of T cell autoimmunity among b-cell antigens in diabetes-prone NOD mice.
Journal of Immunology. 2006; 176: 2654-2661.
49.Jaeckel E, Lipes MA, von Boehmer H. Antigen-specific foxp3-transduced t-cells can control established type 1 diabetes.
Nature Immunol. 2004;5:1028-1035.
50.Lieberman SM, Evans AM, Han B, et al. Identification of the beta cell antigen.
Proc Natl Acad Sci U S A. 2003; 100:8384-8388.
51.Arif S, Timothy I. Tree1 TI, , Thomas P. Astill TP, et al. Autoreactive T cell responses show proinflammatory polarization in diabetes but a regulatory phenotype in health.
J. Clin. Invest. 2004;113:451-463.
52.Kent SC, Chen Y, et al. Expanded T cells from pancreatic lymph nodes of type 1 diabetic subjects recognize an insulin epitope.
Nature. 2005;435:224-228.
53.Rotimi CN, Chen G, Adeyemo AA. A genome-wide search for type 2 diabetes susceptibility genes in West Africans: the Africa America Diabetes Mellitus (AADM) study.
Diabetes. 2004:53:1404.
54.Memon RA, Bessman SP, Mohan C. Impaired mitochondrial metabolism and reduced amphibolic Krebs cycle activity in diabetic rat hepatocytes.
Biochem Mol Biol Int. 1995;6:1079-1089.
55.Hotta K et al. Circulating concentrations of the adipocyte protein adiponectin are decreased in parallel with reduced insulin sensitivity during the progression to type 2 diabetes in rhesus monkeys.
Diabetes. 2001;50:1126–1133.
56.Giroix MH, Rasschaert J, Sener A, et al. Study of hexose transport, glycerol phosphate shuttle and Krebs cycle in islets of adult rats injected with streptozotocin during the neonatal period.
Mol Cell Endocrinol. 1992;83:95-104.
57.Rosen, E. D. et al. PPAR is required for the differentiation of adipose tissue in vivo and in vitro.
Mol. Cell. 1999;4:611–617.
58.La Selva M, Beltramo E, Pagnozzi F, et al. Thiamine corrects delayed replication and decreases production of lactate and advanced glycation end-products in bovine retinal and human umbilical vein endothelial cells cultured under high glucose conditions.
Diabetologia. 1997;40:741-742.
59.Sullivan KA, Feldman EL. New developments in diabetic neuropathy.
Curr Opin Neurol. 2005;18:586-590.
60.Xie XM, Yang ZW, Chen MF. Effects of advanced glycation endproducts on the activity of NF-kappaB and the expression of fibronectin mRNA in the endothelial cells in aged rats.
Zhong Nan Da Xue Xue Bao Yi Xue Ban. 2006;31:883-887.
61.Després J-P, Lemieux I. Abdominal obesity and metabolic syndrome.
Nature. 2006;444: 881-887.
62.Ali M. Integrative Cardiology and Chelation Therapies: The Oxidative-Dysoxygenative Model and Chelation Therapies.
Principles and Practice of Integrative Medicine 6. 2nd ed. New York: Canary 21 Press; 2006.
63.Ali M. Oxygen governs the inflammatory response and adjudicates the man-microbe conflicts.
Townsend Letter for Doctors and Patients. 2005;262:98-103.
64.Ali M. Under Darwin’s Glow [editorial].
J Integrative Medicine. 1999. 3:1
65. Ali M. Darwin, fatigue, and fibromyalgia.
J Integrative Medicine. 1999;3:5-10.
66.Ali M. Darwin, oxidosis, dysoxygenosis, and integration.
J Integrative Medicine. 1999;3:11-16.
67.Ali M.
The Ghoraa and Limbic Exercise. Denville, New Jersey: Life Span Books; 1993.
68.Turnbaugh.PJ, Ley RU, Mahowald MA, et al. An obesity-related gut microbiome with increased capacity for energy harvest.
Nature. 2006;444:1027-1031.
69.Ley RE, Turnbaugh PJ, Klein S, et al. Human gut microbes associated with obesity.
Nature. 2006;444:1022.
70.Bajzer M, Seeley RJ. Obesity and gut flora.
Nature. 2006;444:1009-1010.
71.Ali M. Hurt human habitat and energy deficit – healing through the restoration of krebs cycle chemistry.
Townsend Letter. October 2006:112-116.
72.Ali M. Integrative Nutritional Medicine: Nutrition Seen Through the Prism of Oxygen Homeostasis.
Principles and Practice of Integrative Medicine 5. 2nd ed. New York: Canary 21 Press; 2005.
73.Ali M. Darwin, Dysox, and Disease.
The Principles and Practice of Integrative Medicine 11. New York: Canary 21 Press; 2002.


Dr. Ali’s Nutrient and Herbal Protocols for Preventing and Reversing Diabetes

Majid Ali, M.D.


Suite 1 C, 344 Prospect Ave, Hackensack, NJ 07601

Below are the nutrient, herbal, and spice protocols  formulated by Dr. Ali and prescribed for his patients  for over 30 years. Dr. Ali for the prevention, reversal, or control of diabetes. He highly recommends that they be used under a clinician’s supervision. A special precaution suggested for the clinicians is to consider blood creatinine level of the patient as an indicator of kidney function when prescribing protocols containing potassium, although the potassium doses included are modest.

Two Most Important Questions

Is Diabetes a Sugar Problem?

The Answer: NO.

Is Diabetes An Insulin Problem?

The Answer: Yes. Yes.

Unless specified otherwise, the word at this web site is used for Type 2 diabetes.


1.   If you think, diabetes is a sugar problem, tests done for blood sugar levels for screening for diabetes will be misleading most of the time.
2.   The diagnosis of diabetes will be delayed for five, ten, or more years.
3.   If you are overweight, it will be much more difficult to lose weight. 
4.   Unless you are at your optimal weight, undetected insulin toxicity will injure all your body organs to varying degrees until diabetes is diagnosed and treated for years, usually five to ten or more years.

Important Links
Dr. Ali’s Free Diabetes Course
Diet for Preventing, reversing, and controlling diabetes
Special Recipes for Preventing, reversing, and controlling diabetes

Diabet Protocol

 for Prevention, Reversal, and Control of Diabetes
Two Capsules on Alternate Days
Component /Ingredient
Daily Dose
How It Works
100 mcg
For regulating carbohydrate metabolism, and  controlling blood glucose, and blood pressure, One of diabetes mineral trio (with Selenium, and Molybdenum),  for carbohydrate metabolism, mineral trio, along with Selenium, and Molybdenum, 
Gymnema sylvestre
750 mg
Curbs appetite, Reduces craving for sweets, Increases Energy, Quick recovery after physical activity
Neem extract
50 mg
All-purpose diabetes prevention and reversal
Huckleberry leaf
100 mg
Preserves kidney health and for all-purpose diabetes prevention and reversal
Vanadyl sulfate
20 mg
Complements Gymnema sylvestre

Vascular Protocol

 for Prevention, Reversal, and Control of Vascular Complications of Diabetes. One capsule Twice Daily
Component /Ingredient
How It Works
100 mg
Cardioprotective, enhances heart strenth
100 mg
Cardioprotective, enhances heart strenth
50 mcg
Vascular health
Hawthorne Pyridoxine HCL
200 mg
4:1 Extract
For normalizing blood pressure, anxiety control
300 mg
For blood health and circulation
Pyridoxine HCL
For protein and amino acid metabolism
Vit B6 (as pyridoxine HCL)
Protein metabolism,
Folic acid
800 mcg
Vitamin B 12
1000 mcg
200 mg
Antinflammatory, precents microclot formation in circulating blood
For anxiety associated with prediabetes or diabetes, Howthorne can used as tincture and combined with tincture of assionflowert, seven drops each added to once of cold water and sipped dlowly. This combination can be repeated three times inn24 hours, if needed.

K-Mag-Tau Diabetes Protocol

 for Bowel Detox In Prevention, Reversal, and Control of Diabetes

One Tablet Twice daily

Component /Ingredient
In Mgs
How It Works
Highly recommended for their complementary benefits of bowel-blood detox for diabetes, circulation, and heart health
Oral Chelation Protocol for Diabetes and Heart
 for Prevention, Reversal, and Control of Diabetes, Stroke, and Diabetic Heart and Vascular Complications
One Tablet Twice daily
Component /Ingredient
In Mgs
How It Works
1000 mg
All ingredient produce their benefits by their integrated roles in cleansing blood, preventing stickiness of blood cells, preserving endo cells (endothelial lining the inside of blood vessels. ner (endo cells)
250 mg
25 mg
250 mg
Vitamin C

Turmeric-Vitamin C Protocol

 for Prevention, Reversal, and Control of Diabetes
Twice Daily, Take three Times A Day If Any infection coexist
Component /Ingrediant
In Mgs
How It Works
1.3 teaspoon
Cleanses blood, improves circulation, reduces stickiness of blood cells
Vitamin C
1000 mg
Antioxidant, Improves Blood Circulation Blood cleanser

Blood Cells Tell The Insulin Toxicity Story

Healthy Blood Cells for Comparative Study. Figure 1
Early Stress on Red Blood Cells (lower picture) . Figure 2

Microplaques in Circulating Blood

When Blood Glucose Level Rises Above 200 mg/dL

Figure 13 (top) and figure 14 (bottom) show two microplaques in a patient who had received three unsuccessful angioplasties for advanced IHD. Photomicrographs were taken the day after a major nosebleed. Note the compaction of necrotic debris and blood elements in microplaques as contrasted with loose structure of microclots in figure 11.

Red Blood Cells in a Micro-clot In Uncontrolled Diabetes (upper Picture) Figure 3
Red Blood Cell Clot Breaking Up (lower Picture) Figure 4

Micro-plaque Formation In Uncontrolled Diabetes (both pictures) Figures 5-6

Figure 7 (top) illustrates severely damaged erythrocytes in a 52-year-old man with persistent atrial fibrillation. Close examination shows some zones of congealing surrounding many damaged red blood cells.
Dr. Ali’s Video Library

How Do You Reverse Diabetes Majid Ali MD on Vimeo › Majid Ali › Videos
May 24, 2014 – Uploaded by Majid Ali
Professor Majid Ali shares information about “How Do You Reverse Diabetes

How Do You Reverse Diabetes Majid Ali MD on Vimeo › Majid Ali › Videos
May 24, 2014 – Uploaded by Majid Ali
Professor Majid Ali shares information about “How Do You Reverse Diabetes
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What Is Your Diabetes Subtype? Majid Ali MD on Vimeo › Majid Ali › Videos
May 19, 2014 – Uploaded by Majid Ali
I recognize two subtypes of diabetes Type 2: diabetes Type 2A (high insulin) anddiabetes Type2 B (insulin …

Vegetarian Diet for Type 2 Diabetes Majid Ali MD on Vimeo › Majid Ali › Videos
Apr 23, 2014 – Uploaded by Majid Ali
Professor Majid Ali shares information about “Vegetarian Diet for … Vegetarian Diet for Type 2 Diabetes Majid …
Can I Have Diabetes With Normal A1c Test Majid Ali MD

My Top Three Weight Loss and Anti-diabetes Omelettes Majid Ali MD

Two Most Important Questions After Diabetes Diagnosis Majid Ali MD

Diabetes and insulin Majid Ali MD

How Do I Reverse Diabetes Majid Ali MD

Diabetes and Insulin Library

Majid Ali, M.D.

A Comprehensive Library of the Science and Philosophy of Holism For Preventing and Reversing Diabetes 


Diabetes Reversal With Insulin Detox | Ali Diabetes

Jun 4, 2017 – Dr. Ali’s Breakfast for Losing Weight, Reversing Diabetes, and Staying … ….. Apr 18, 2017Majid Ali, M.D. Dr. Ali’s Insulin Homeostasis Course …

Diabetes Is Rooted In Insulin Toxicity – Part Two | Ali Diabetes

Jun 4, 2017 – Majid Ali, M.D. Diabetes Begins 15–20 years before it is diagnosed Text Reproduced … Dr.Ali’s Breakfast for Losing Weight, Reversing Diabetes, and Staying … …

Oxidation Model of Diabetes Validated – 20 Years Later | Ali Diabetes

Jun 5, 2017 – Majid Ali, M.D. I published my oxidative model of diabetes in 1995. … Oct 31, 2014 – Dr.Ali’s Breakfast I consider an optimal breakfast to be the first essential in any … …

Dr’s Insulin Course | Ali Diabetes

May 31, 2017 – Apr 23 … Apr 18, 2017Majid Ali, M.D. Dr. Ali’s Insulin Homeostasis Course Insulin | The Ali Academy Community ….. Dr. Ali’sBest Anti-Insulin Toxicity Breakfast.

Dr. Ali’s Start-Low-Build-Slow Principle | Ali Diabetes

Majid Ali, M.D. Dr. Ali’s Start-Low-Build-Slow Principle In all guidelines for natural remedies for … Posted on April 24, 2017 by Majid Ali MD Leave a comment.

Dr Ali Breakfast videos part 1 | The Ali Academy Community

Dr. Ali, Your Breakfast Is Great!! Majid Ali MD An Excellent Anti-diabetes Breakfast Majid Ali MD Lecithin for Dr. Ali’s Breakfast Majid Ali MD More Energy With Dr.

Dr. Ali’s Insulin Reduction Protocol

Majid Ali, M.D. … Concurrent Reduction of Blood Insulin and Blood Sugar Levels With Dr. Ali’s Insulin Reduction Protocol in a 58-Yr-Old Woman … Please consider “Dr. Ali’s Breakfast” (protein shake described below) four or five days a week.




Dr. Ali’s Breakfast 2017 | – Who Is Dr. Ali?

Oct 8, 2016 – Majid Ali, M.D. Majid Ali, M.D. Dr. Ali’s Breakfasts Are Ideas, Not Products We Value Your Suggestions. Please Send Them to …

Dr. Ali’s Breakfast 2017 | – Who Is Dr. Ali?

Oct 8, 2016 – Posts about Dr. Ali’s Breakfast 2017 written by Majid Ali MD.

Dr. Ali’s Insulin-Wise Breakfast – Dr. Ali’s Virtual Medical Library’s_breakfast.htm

Indeed, next to the subject of chronic anger I devote more time energy—and energy— to the matter of “Dr. Ali’s breakfast” than any other subject. In this chapter, I …

Majid Ali MD, Dr. Ali’s 5- Spices Yogurt Breakfast for Reversing … › Majid Ali › Videos
Oct 30, 2014

I describe my 5 Spices Yogurt Breakfast and recommend its inclusion in my Insulin-Monitored Diabetes …

Weight Loss With Dr. Ali’s Breakfast Majid Ali MD on Vimeo › Majid Ali › Videos
Apr 23, 2014

Professor Majid Ali shares information about “Weight Loss With Dr. Ali’s Breakfast

Will Dr. Ali’s Breakfast Reverse Heart Disease Majid Ali MD on Vimeo › Majid Ali › Videos
Apr 24, 2014

Professor Majid Ali shares information about “Will Dr. Ali’s Breakfast Reverse Heart Disease”

Fish Oil for Dr. Ali’s Breakfast Majid Ali MD on Vimeo › Majid Ali › Videos
May 12, 2014

Professor Majid Ali shares information about “Fish Oil for Dr. Ali’s Breakfast

Lecithin for Dr. Ali’s Breakfast Majid Ali MD on Vimeo › Majid Ali › Videos
Apr 19, 2014

Professor Majid Ali shares information about “Lecithin for Dr. Ali’s Breakfast

Majid Ali MD, Dr. Ali’s Spicy Anti-Diabetes Breakfasts on Vimeo › Majid Ali › Videos
Jan 28, 2015

Please try my spice breakfast suggestions. I don’t think they will disappoint you.


Posted on April 23, 2017 by Majid Ali MD Leave a comment. Majid AliDr Ali’s Best Omelette Recipes for Weight loss and Preventing and Reversing Diabetes.


Reversing Prediabetes and Diabetes With 3D Plan: Insulin-Wise and Insulin-Unwise Foods and Meals

Majid Ali, M.D.

A Question-Answer Learning Exercise of Great Importance for Reversing Hyperinsulinism (Prediabetes) and Reversing Diabetes


Reversing Prediabetes and Diabetes 3D Plan


Three prongs of the Reversing Diabetes D3 are:

                                     D  for diet

                                     D  for Detox

                                     D  for Dysox

I use the term dysox in the context of controlling hyperinsulinism and reversing Type 2 diabetes (T2D) for oxygen-related co-morbidities of both hyperinsulinism and T2D.


Questions Exercise

For Losing Weight and Reversing Prediabetes (Hyperinsulinism) and Diabetes

with Diabetes 3D

I suggest that you print these questions and circle your answer in light of materials presented at this website. Specifically, which one of the following food/meal options are insulin-wise compared with others. The answers and explanatory comments appear at the end of questions.

  1. Popcorn in the movie house or a slice of pizza before the movie.
  2. Popcorn with butter or popcorn without butter
  3. Whole milk yogurt or fat-free milk
  4. Yogurt or kefer for snack
  5. Fruit juice or protein shake
  6. Egg omelette or scrambled egg
  7. Fish meal or beef meal
  8. Whole milk or 2% milk
  9. Toast with olive oil or with it
  10. Blueberries snack with or without cheese
  11. Peanut butter snack or an apple
  12. Foods that cause sugar spikes or foods that trigger insulin spikes
  13. Salad with olive or salad with salad dressing
  14. Flaxseed oil or freshly ground flaxseeds
  15. An apple in the afternoon at work or two teaspoons of almond butter
  16. Breakfast Protein Shake with or without Spices
  17. White rice of brown rice for dinner
  18. For high morning blood sugar levels due to diabetes, four ounces of protein shake in the morning before breakfast or at bed time
  19.  Carrots or radishes in equal weight  
  20. Total gluten abstinence for GERD-gastritis complex or for colitis
  21. Aspartame or saccharine as sweeteners 


Essentials of Relationships Between Food Spikes and Insulin Spikes

  1. Every food that causes a sugar spike triggers an insulin spike.
  2. Every insulin spike causes a sharp drop in blood sugar level (hypoglycemia).
  3. Every sharp drop in blood sugar level creates a craving for foods that produce sugar spikes.
  4. Every sharp insulin spike sets the stage for secondary, albeit lower, insulin spikes.
  5. The larger the number of insulin spikes, higher the chance of developing hyperinsulinism (insulin toxicity by its more appropriate name)  


Plant Remedies and Supplements

for Reversing Hyperinsulinism (Prediabetes) and Diabetes

Phytofactors (Plant Remedies) 

  1. Aloe Vera
  2. Bitter melon
  3. Cinnamon
  4. Fenugreek
  5. Flaxseed
  6. Garlic
  7. Ginseng (Panax ginseng)
  8. Gymnema sylvestre
  9. Huckelberry
  10. Neem
  11. Nopal (prickly pear cactus)

Nutritional Supplements

  1. Coenzyme Q10
  2. Chromium
  3. Lipoic acid (alpha lipoic) 
  4. Magnesium, potassium, Taurine
  5. Cinnamon                    
  6. Chromium                   
  7. Vanadyl sulfate           
  8. Multivitamin
  9. Multimineral


Highly recommended readings at this website

  1. Reversing diabetes 3D
  2. Shifting Focus from Glycemic Status to Insulin Homeostasis for stemming Global tides of hyperinsulinism and Type 2 Diabetes
  3. Free Access Dr. Ali’s Insulin Course
  4. Free Access Dr. Ali’s Library of Videos and Articles


Video and Article Library

Majid Ali, M.D. * Insulin Toxicity De-mystifies the Metabolic Syndrome …

Jun 28, 2012 – Uploaded by majid ali

The true mature of the metabolic syndrome is insulin toxicity. The term metabolic syndrome creates creates …

Majid Ali MD, Dr. Ali’s Book on Reversing Diabetes – Dr. Ali’s Plan for … › Majid Ali › Videos
Nov 6, 2014

I outline the contents of this book on reversing diabetes Type 2. I explain how it begins with insulin toxicity …

How Do You Reverse Diabetes Majid Ali MD on Vimeo › Majid Ali › Videos
May 24, 2014

Professor Majid Ali shares information about “How Do You Reverse Diabetes

Majid Ali MD, Castor Oil Rubs for Insulin Detox for Weight Loss and … › Majid Ali › Videos
Jan 24, 2015

Type 2 diabetes is an insulin-toxicity state for years before the body reserves ofinsulin are depleted and the …

Dr. Ali’s Insulin Reduction Protocol

Majid Ali, M.D. … For individuals with pre-diabetes with insulin toxicity but without high blood sugar levels, my … I present this subject at length in my book entitled “Dr. Ali’s Plan for Reversing Diabetes” and in a 40-minute video seminar that can …

Dr. Ali’s Diabetes Reversal program – Dr. Ali’s Virtual Medical Library’s_diabetes_reversal_guidelines.htm

Majid Ali, M.D.. The science and philosophy of reversing diabetes Type 2 associated with excess insulinis simple to understand. On this website and in my video …

Reversing Diabetes – Majid Ali

Simply stated, diabetes Type 2A is a state of insulin toxicity created by insulin … 1 of a video seminar on reversing diabetes, Professor Majid Ali, M.D. presents a …

Dr. Ali’s Insulin-Based Diabetes Reversal | –

Jan 11, 2017 – Majid Ali, M.D. Workable Simplicity to Understand Maddening Complexity of Three Scourges of Our Time Dr. Ali’s InsulinDiabetes Video and …


Diabetes Reversal With Insulin Profiling : Dr Majid Ali : Free Download …

May 5, 2014 – For more information on the subject , please consider Prof.Ali`s Video seminars and courses available as downloads at

Insulin Toxicity | Childrens Health Corps

Jul 15, 2014 – Insulin Toxicity Video Series … Insulin-Wise Eating Majid Ali, M.D. To reverse pre-diabetesand diabetes (completely or partially), my primary …

Majid Ali MD – Home | Facebook

… about this. Majid Ali, MD has compiled a free video library of thousands of videos pertaining to health. … Ozone | Diabetic or Insulin Neuropathy. Ozone by …

THESE ARE VIDEO DOWNLOADS from … Instant Seminars with Majid Ali, M.D.. ThevideosInsulin Toxicity and Reversal of Diabetes. Skin.

Gut Fermentation Control


Majid Ali, M.D.


A Simplicity for All Health and Healing Matters

All Diseases Begin and Persist With Cellular Fermentation and their treatment must include control of fermentation in the body and the mind.

 What Is Fermentation?

Fermentation is a sequences  of natural chemical reactions in which cells produce alcohols and acids because there is not enough oxygen to produce sand utilize clean ATP cellular energy. So, fermentation fundamentally is a problem of deficit of functional oxygen


Damaging Effects of Fermentation

In the early 199os,  I introduced the term dysox for a state in which oxygen fails to drive and govern the generation and utilization of energy in the human cells. I recognized this state as the beginning of cellular dysfunction / dis-ease / and disease.

In 2004, the results of my first study were published in the journal Townsend Letter.

In 2015, the results of much larger study were published in the journal Nature.


The Seven for Fermentation Control

1. Dr. Ali’s Breakfasts (Ideas not products)

2. Slow Limbic Breathing

3. Insulin-Smart Foods

4. Insulin-Wise Living

5. Purpose in Life

6. Freedom from Hate

&. Love

Suggested Videos and Articles for Love and Freedom

1. Our Shared African Grandmothers

2. The Pre-East Way for Health and Healing

What Causes Fermentation in the Head?

1. Recycling past misery

2. Not learning and practicing compassion

3. Hate (To hate anyone, one must first hate oneself)

4. Denying hope

5. Not being kind to the body so the body can be kind.

6. Not breathing slow, speaking softy, respecting food

7. Not being oxygen-wise and insulin-intelligent

See Videos on the Subjects at the End


The Sun-Soil Model of

 Health and Healing


The Sun-Soil Model of Health and Healing

Oxygen rules the body from its three-legged throne.

Library of Videos

See Dr. Ali’s Recipes

See Dr. Ali’s Peer-reviewed publications at

For Dr. Ali’s Books, Please Go To

For more at



Reversing Diabetes – Seven Simple Lessons

Insulin-Smart Recipes


Insulin Video Series



Link to Library of Recipes for Reversing Diabetes and Insulin Detox

Videos and Read More on Reversing Diabetes At This Web Site. Use the search box to access Diabetes Course.

Who Is Doctor Ali?

Insulin-Toxic Obesity
Fats Are Not Fattening Americans
 Demand Nor Offer Forgiveness, Act Forgiveness

What Is the Most Important Question in Science, Health, and Healing
What Is the Second Most Important Question in Science, Health, and Healing

57 Percent Black American Women Are Obese

 Majid Ali, M.D.

Why are more black American women obese than American women of other ethnicity? 

National Health and Nutrition Examination Survey (NHANES) releases federal health data every two years. It is considered to be the gold standard in national stats. In November, NHANES reported that 57 percent of black American women were obese. In sharp contrast, only 12% of Asians were obese.

Why are more black American women obese?

My simple answer: They are more toxic. What makes black American women more toxic? Answer: toxic foods, toxic environments, toxic thoughts (frustration, anger, and fear). How do I know that? Because obesity is toxicity. It is this simple.

A Calorie Is a Calorie

The New York Times once asserted that a calorie is a calorie. It is hard to believe that any Times’ writer can be that ignorant. But it should really not surprise anyone. In matters of nutrition, the Times only seeks the opinions of those doctors who do not practice nutritional medicine. on matters of nutrition.

I point out that according to NHANES, the rate of obesity in black American women continued to rise in spite of falling consumption of full-calorie soda and calorie intake in recent years. Ironically, this was recognized in the Times’ article.

Obesity Is Toxicity

For a full discussion of this subject, please consider my three FREE courses listed below.

  1. Dr. Ali’s Obesity Course
  2. Dr. Ali’s Insulin Course
  3. Dr. Ali’s Fermentation Course